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Targeting androgen receptor (AR) with antiandrogen Enzalutamide increases prostate cancer cell invasion yet decreases bladder cancer cell invasion via differentially altering the AR/circRNA-ARC1/miR-125b-2-3p or miR-4736/PPARγ/MMP-9 signals

机译：靶向雄激素受体（AR）与抗抗原醛酰胺酰胺增加，通过差异地改变AR / CircRNA-Arc1 / miR-125B-2-3P或MIR-4736 /PPARγ/ MMP-9信号，降低前列腺癌细胞侵袭且膀胱癌细胞侵袭降低

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Gang Deng; Ronghao Wang; Yin Sun; Chi-Ping Huang; Shuyuan Yeh; Bosen You; Changyong Feng; Gonghui Li; Shenglin Ma; Chawnshang Chang;
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1. Targeting androgen receptor (AR) with antiandrogen Enzalutamide increases prostate cancer cell invasion yet decreases bladder cancer cell invasion via differentially altering the AR/circRNA-ARC1/miR-125b-2-3p or miR-4736/PPAR gamma/MMP-9 signals [J] . Deng Gang, Wang Ronghao, Sun Yin, Cell death and differentiation . 2021,第7期

机译：靶向雄激素受体（AR）具有抗抗原醛酰胺酰胺，增加前列腺癌细胞侵袭且通过差异地改变AR / CircRNA-ARC1 / miR-125B-2-3P或MIR-4736 / PPAR伽马/ MMP-9信号来降低膀胱癌细胞侵袭
2. Anti-androgen enzalutamide enhances prostate cancer neuroendocrine (NE) differentiation via altering the infiltrated mast cells?→?androgen receptor (AR)?→?miRNA32 signals [J] . Qiang Dang, Lei Li, Hongjun Xie, Molecular oncology. . 2015,第7期

机译：抗雄激素恩杂鲁胺通过改变浸润的肥大细胞增强前列腺癌神经内分泌（NE）的分化→→→雄激素受体（AR）→→→miRNA32信号
3. Targeting AR-Beclin 1 complex-modulated growth factor signaling increases the antiandrogen Enzalutamide sensitivity to better suppress the castration-resistant prostate cancer growth [J] . Zhang Meng, Sun Yin, Meng Jialin, Cancer letters . 2019,第期

机译：靶向Ar-Beclin1复合调制的生长因子信号传导增加了抗抗原醛酰胺敏感性，以更好地抑制抗阉割的前列腺癌生长
4. Role of the Androgen Receptor and PI3K/Akt in the Survival of Androgen-Refractory Prostate Cancer Cells [C] . Haojie Huang, Donald J. Tindal International Symposium on Hormonal Carcinogenesis . 2005

机译：雄激素受体和PI3K / AKT在雄激素难治性前列腺癌细胞存活中的作用
5. MED19 Alters Genomic Occupancy of the Androgen Receptor and Gene Expression in Prostate Cancer Cells, Driving Growth under Low Androgen [D] . Weber, Hannah. 2020

机译：Med19改变了前列腺癌细胞中雄激素受体和基因表达的基因组占据，在低雄激素下促进生长
6. Targeting androgen receptor (AR) with antiandrogen Enzalutamide increases prostate cancer cell invasion yet decreases bladder cancer cell invasion via differentially altering the AR/circRNA-ARC1/miR-125b-2-3p or miR-4736/PPARγ/MMP-9 signals [O] . Gang Deng, Ronghao Wang, Yin Sun, 2021

机译：靶向雄激素受体（AR）与抗抗原醛酰胺酰胺增加通过差异地改变AR / CircRNA-Arc1 / miR-125B-2-3P或MIR-4736 /PPARγ/ MMP-9信号降低前列腺癌细胞侵袭且膀胱癌细胞侵袭降低
7. Infiltrating mast cells enhance prostate cancer invasion via altering LncRNA-HOTAIR/PRC2-androgen receptor (AR)-MMP9 signals and increased stem/progenitor cell population [O] . Lei Li, Qiang Dang, Hongjun Xie, 2015

机译：渗透肥大细胞通过改变LNCRNA-HOTAIR / PRC2-andRogen受体（AR）-MMP9信号和增加的茎/祖细胞群，增强前列腺癌侵袭
8. Molecular Determinants of Prostate Cancer Progression Across Race- Ethnicity. Project A - The Human 5RD5A2 Gene and Prostate Cancer Progression. Project B - Androgen Receptor (AR) Signaling in Prostate Cancer Progression. Project C - Cellular and Molecular Markers of Prostate Cancer Progression Core - Epidemiology Core [R] . Ross, R. R. , Reichardt, J. , Coetzee, G. A. , 2002

机译：跨越种族的前列腺癌进展的分子决定因素。项目a - 人类5RD5a2基因和前列腺癌进展。项目B - 雄激素受体（aR）在前列腺癌进展中的信号传导。项目C - 前列腺癌进展核心的细胞和分子标记 - 流行病学核心

Targeting androgen receptor (AR) with antiandrogen Enzalutamide increases prostate cancer cell invasion yet decreases bladder cancer cell invasion via differentially altering the AR/circRNA-ARC1/miR-125b-2-3p or miR-4736/PPARγ/MMP-9 signals

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