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机译:Saikosaponin D通过PI3K / AKT / MTOR途径抑制胰腺星状细胞的自噬改善胰腺纤维化
Tianjin Nankai Hosp Inst Acute Abdominal Dis Integrated Tradit Chines Dept Cell &
Mol Biol;
Tianjin Nankai Hosp Inst Acute Abdominal Dis Integrated Tradit Chines Dept Cell &
Mol Biol;
Tianjin Nankai Hosp Inst Acute Abdominal Dis Integrated Tradit Chines Dept Cell &
Mol Biol;
Tianjin Nankai Hosp Inst Acute Abdominal Dis Integrated Tradit Chines Dept Cell &
Mol Biol;
Tianjin Nankai Hosp Dept Hepatobiliary &
Pancreat Surg Tianjin 300100 Peoples R China;
Tianjin Nankai Hosp Inst Acute Abdominal Dis Integrated Tradit Chines Dept Cell &
Mol Biol;
Saikosaponin d; Pancreatic fibrosis; Pancreatic stellate cells; Autophagy; PI3K/Akt/mTOR; TGR beta-1/Smads;
机译:Saikosaponin D通过PI3K / AKT / MTOR途径抑制胰腺星状细胞的自噬改善胰腺纤维化
机译:Mitofusin2通过抑制PI3K / Akt / mTOR信号通路诱导胰腺癌细胞自噬
机译:囊性纤维化跨膜电导调节剂通过抑制小鼠的PI3K / AKT / MTOR途径来改善脂多糖诱导的急性肺损伤
机译:胰腺星状细胞活化伴有胰腺星状细胞活化,伴有秘密的变化
机译:多靶点受体酪氨酸激酶抑制剂利尼法尼(ABT-869)通过磷酸肌醇3激酶(PI3K)/ AKT依赖性信号传导途径诱导凋亡并抑制白血病细胞的增殖。
机译:辅酶Q10通过PI3K / AKT / mTOR信号通路抑制胰腺星状细胞的活化
机译:维甲酸通过抑制Wnt /β-Catenin信号通路改善胰腺纤维化并抑制实验性慢性胰腺炎小鼠胰腺星状细胞的活化。