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机译:通过通过AMPK-MTOR信号传导途径增强自噬,Pioglitazone通过增强自噬来保护管状细胞免受缺氧/雷诺损伤
Nanchang Univ Dept Urol Affiliated Hosp 2 1 Minde Rd Nanchang 330006 Jiangxi Peoples R China;
Nanchang Univ Affiliated Hosp 4 Dept Endocrinol Nanchang Jiangxi Peoples R China;
Nanchang Univ Dept Urol Affiliated Hosp 2 1 Minde Rd Nanchang 330006 Jiangxi Peoples R China;
Nanchang Univ Dept Urol Affiliated Hosp 2 1 Minde Rd Nanchang 330006 Jiangxi Peoples R China;
Nanchang Univ Dept Urol Affiliated Hosp 2 1 Minde Rd Nanchang 330006 Jiangxi Peoples R China;
Nanchang Univ Dept Urol Affiliated Hosp 2 1 Minde Rd Nanchang 330006 Jiangxi Peoples R China;
Pioglitazone; Autophagy; Tubular cells; Hypoxia/reoxygenation; AMP;
机译:通过通过AMPK-MTOR信号传导途径增强自噬,Pioglitazone通过增强自噬来保护管状细胞免受缺氧/雷诺损伤
机译:Schisantherina通过激活PI3K / AKT信号通路来保护肾小管上皮细胞免受缺氧/雷诺损伤的影响
机译:外源MSC通过JAK / STAT信号通路介导的HMGB1调节减轻肾小管上皮细胞的缺氧/复氧损伤
机译:DHHP-6保护H9C2心肌细胞免受缺氧/雷诺诱导的氧化损伤
机译:心肌细胞缺氧酸中毒和缺氧复氧中的凋亡信号通路。
机译:Angptl2的敲低通过抑制TLR4 / NF-κB信号传导途径和激活NRF2 / HO-1信号通路保护肾小管上皮细胞免受缺氧/雷诺诱导的损伤
机译:Angptl2的敲低通过抑制TLR4 / NF-κB信号传导途径和激活NRF2 / HO-1信号通路,保护肾小管上皮细胞免受缺氧/雷诺诱导的损伤