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机译:Adam17的敲低抑制细胞增殖并通过EGFR-PI3K-AKT激活增加HCT-8结直肠癌中的奥沙利铂敏感性
North China Univ Sci &
Technol Affiliated Hosp Dept Gen Surg Tangshan Hebei Peoples R China;
North China Univ Sci &
Technol Affiliated Hosp Dept Gen Surg Tangshan Hebei Peoples R China;
North China Univ Sci &
Technol Med Expt Res Ctr Tangshan Hebei Peoples R China;
North China Univ Sci &
Technol Affiliated Hosp Dept Gen Surg Tangshan Hebei Peoples R China;
North China Univ Sci &
Technol Affiliated Hosp Dept Gen Surg Tangshan Hebei Peoples R China;
North China Univ Sci &
Technol Affiliated Hosp Dept Gen Surg Tangshan Hebei Peoples R China;
ADAM17; EGFR/PI3K/AKT signaling pathway; Colorectal cancer; Chemoresistance;
机译:Adam17的敲低抑制细胞增殖并通过EGFR-PI3K-AKT激活增加HCT-8结直肠癌中的奥沙利铂敏感性
机译:敲低KLK11通过抑制结直肠癌细胞的增殖和通过抑制PI3K / AKT信号通路激活凋亡来逆转奥沙利铂耐药性
机译:敲低KLK11通过抑制结直肠癌细胞的增殖和通过抑制PI3K / AKT信号通路激活凋亡来逆转奥沙利铂耐药性
机译:将正常的乳腺上皮线粒体引入MCF-7人乳腺癌细胞系可抑制增殖并增加药物敏感性
机译:Caspase-3促进细胞增殖并抑制大肠癌中DNA损伤诱导的坏死。
机译:敲低KLK11抑制人结肠直肠癌的细胞增殖并提高奥沙利铂敏感性
机译:击倒MMP-7可抑制细胞增殖并增强对结肠癌细胞中5-氟尿嘧啶和X射线辐射的敏感性