机译:靶向PAK1与小分子药物AK963 / 40708899通过下调PAK1活动和PAK1相关信号通路的下调抑制胃癌细胞增殖和侵袭
Department of Cell Biology Key Laboratory of Cell Biology Ministry of Public Health and Key;
Department of Cell Biology Key Laboratory of Cell Biology Ministry of Public Health and Key;
Key Laboratory of Structure‐Based Drug Design and Discovery of Ministry of EducationShenyang;
Key Laboratory of Structure‐Based Drug Design and Discovery of Ministry of EducationShenyang;
Key Laboratory of Structure‐Based Drug Design and Discovery of Ministry of EducationShenyang;
Department of Cell Biology Key Laboratory of Cell Biology Ministry of Public Health and Key;
AK963/40708899; PAK1; inhibitor; gastric cancer;
机译:靶向PAK1与小分子药物AK963 / 40708899通过下调PAK1活动和PAK1相关信号通路的下调抑制胃癌细胞增殖和侵袭
机译:用小分子药物AZA197靶向Cdc42可通过下调PAK1活性抑制临床前小鼠异种移植模型中的原发性结肠癌生长并延长生存期
机译:rugogdi2通过Rac1 / Pak1 / Limk1途径抑制人胃癌细胞增殖,侵袭和耐药性
机译:Hergl频道在人胃癌中过表达,其活性调节细胞增殖:一种新的预后和治疗靶标?
机译:在乳腺癌1(DBC1)中删除,通过负调控替代NFkappaB信号通路抑制B细胞激活。
机译:通过下调PAK1活性用小分子药物AZA197靶向Cdc42可抑制原发性结肠癌的生长并延长临床前小鼠异种移植模型的存活时间
机译:靶向PAK1与小分子药物AK963 / 40708899通过下调PAK1活动和PAK1相关信号通路的下调抑制胃癌细胞增殖和侵袭