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机译:羧基末端调节剂蛋白呈正常调节AKT磷酸化,并作为乳腺癌的致癌钳
Institute of Clinical Medicine National Cheng Kung University Medical College No. 35 Siao-dong;
Institute of Clinical Medicine National Cheng Kung University Medical College No. 35 Siao-dong;
Department of Medical Education and Research Kaohsiung Veterans General Hospital Kaohsiung Taiwan;
Institute of Clinical Medicine National Cheng Kung University Medical College No. 35 Siao-dong;
Institute of Clinical Medicine National Cheng Kung University Medical College No. 35 Siao-dong;
Institute of Clinical Medicine National Cheng Kung University Medical College No. 35 Siao-dong;
Department of Surgery Kaohsiung Veterans General Hospital Kaohsiung Taiwan Department of;
Department of Surgery Kaohsiung Veterans General Hospital Kaohsiung Taiwan;
Genomics Research Center Academic Sinica No. 128 Academia Road Taipei 115 Taiwan;
Graduate Institute of Cancer Biology and Drug Discovery College of Medical Science and Technology;
Institute of Clinical Medicine National Cheng Kung University Medical College No. 35 Siao-dong;
Genomics Research Center Academic Sinica No. 128 Academia Road Taipei 115 Taiwan;
Institute of Clinical Medicine National Cheng Kung University Medical College No. 35 Siao-dong;
机译:羧基末端调节剂蛋白呈正常调节AKT磷酸化,并作为乳腺癌的致癌钳
机译:Rack1介导SRC与药物转运蛋白p-糖蛋白的结合,并通过调节乳腺癌细胞中的Caveolin-1磷酸化来调节其活性
机译:四分之一的LIM蛋白1通过抑制人乳腺癌细胞中AKT磷酸化下调雌激素受体α的活性
机译:具有PI3K-AKT路径畸变的患者衍生的乳腺癌异种移植物中AKT同种型的磷酸化位点的平行反应监测测定
机译:Biochanin A和锰可调节HER-2阳性乳腺癌细胞的致癌信号和抗氧化功能。
机译:羧基末端调节蛋白通过调节Akt磷酸化积极发挥头颈部鳞状细胞癌的致癌驱动力。
机译:羧基 - 末端调节剂蛋白通过调节AKT磷酸化,作为头部和颈部鳞状细胞癌中的致癌驱动器
机译:通过磷酸化依赖性调节skp 2稳定性和致癌功能来研究akt1在前列腺癌发展中的作用。