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机译:ALS连锁突变体SOD1通过促进Bcl-2的构象变化来破坏线粒体
Frances and Joseph Weinberg Unit for ALS Research, Farber Institute for Neurosciences, Department of Neuroscience, Thomas Jefferson University, Philadelphia, PA 19107, USA,;
Frances and Joseph Weinberg Unit for ALS Research, Farber Institute for Neurosciences, Department of Neuroscience, Thomas Jefferson University, Philadelphia, PA 19107, USA,;
Frances and Joseph Weinberg Unit for ALS Research, Farber Institute for Neurosciences, Department of Neuroscience, Thomas Jefferson University, Philadelphia, PA 19107, USA,|Neurological Institute IRCCS ‘C. Mondino’, Pavia, Italy and;
Frances and Joseph Weinberg Unit for ALS Research, Farber Institute for Neurosciences, Department of Neuroscience, Thomas Jefferson University, Philadelphia, PA 19107, USA,;
Department of Neurology, University of Massachusetts School of Medicine, Worcester, MA 01655, USA;
Frances and Joseph Weinberg Unit for ALS Resea;
机译:ALS连锁突变体SOD1通过促进Bcl-2的构象变化来破坏线粒体。
机译:抗突变SOD1 / Bcl-2毒性线粒体复合物的小肽可恢复突变SOD1介导的ALS中的线粒体功能和细胞活力
机译:VDAC1和ALS链接的SOD1突变体之间的相互作用:对线粒体健康的影响
机译:通过质谱法研究Als-Linked突变体PFN1蛋白与天然化合物的非共价相互作用
机译:WT和SOD1突变体斑马鱼中Riluzole暴露的生理和行为作用
机译:ALS连锁突变体SOD1通过促进Bcl-2的构象变化来破坏线粒体
机译:ALS连锁突变体SOD1通过促进Bcl-2的构象变化来破坏线粒体