首页> 美国卫生研究院文献>Acta Pharmacologica Sinica >SM905 an artemisinin derivative inhibited NO and pro-inflammatory cytokine production by suppressing MAPK and NF-κB pathways in RAW 264.7 macrophages

【2h】

SM905 an artemisinin derivative inhibited NO and pro-inflammatory cytokine production by suppressing MAPK and NF-κB pathways in RAW 264.7 macrophages

机译：青蒿素衍生物SM905通过抑制RAW 264.7巨噬细胞中的MAPK和NF-κB途径来抑制NO和促炎性细胞因子的产生

代理获取

本网站仅为用户提供外文OA文献查询和代理获取服务，本网站没有原文。下单后我们将采用程序或人工为您竭诚获取高质量的原文，但由于OA文献来源多样且变更频繁，仍可能出现获取不到、文献不完整或与标题不符等情况，如果获取不到我们将提供退款服务。请知悉。

页面导航

摘要
著录项
引文网络
相似文献
相关主题

摘要

Aim:To elucidate the anti-inflammatory potentials and underlying mechanisms of SM905, a novel artemisinin derivative, in lipopolysaccharide (LPS)-stimulated murine macrophage RAW 264.7 cells.

机译：目的：阐明新型青蒿素衍生物SM905在脂多糖（LPS）刺激的鼠巨噬细胞RAW 264.7细胞中的抗炎潜力及其潜在机制。

著录项

期刊名称 Acta Pharmacologica Sinica
作者
Jun-xia Wang; Li-fei Hou; Yang Yang; Wei Tang; Ying Li; Jian-ping Zuo;
展开▼
作者单位

展开▼
年(卷),期 2009(30),10
年度 2009
页码 1428–1435
总页数 8
原文格式 PDF
正文语种
中图分类药学;
关键词
artemisinin SM905 NO iNOS inflammation MAPK NF-κB cyclooxygenase 2 TLR4 receptor;

机译：青蒿素;SM905;NO;iNOS;炎症;MAPK;NF-κB;环氧合酶2;TLR4受体;

相似文献

外文文献
中文文献
专利

1. SM905, an artemisinin derivative, inhibited NO and pro-inflammatory cytokine production by suppressing MAPK and NF-κB pathways in RAW 264.7 macrophages [J] . Jun-xia WANG, Li-fei HOU, Yang YANG, 中国药理学报：英文版 . 2009,第010期

机译：青蒿素衍生物SM905通过抑制RAW 264.7巨噬细胞中的MAPK和NF-κB途径来抑制NO和促炎性细胞因子的产生
2. SM905, an artemisinin derivative, inhibited NO and pro-inflammatory cytokine production by suppressing MAPK and NF-KB pathways in RAW 264.7 macrophages [J] . Jun-xia WANG, Li-fei HOU, Yang YANG, 中国药理学报：英文版 . 2009,第010期

机译：青蒿素衍生物SM905通过抑制RAW 264.7巨噬细胞中的MAPK和NF-KB途径来抑制NO和促炎性细胞因子的产生
3. Recombinant CC16 protein inhibits the production of pro-inflammatory cytokines via NF-κB and p38 MAPK pathways in LPS-activated RAW264.7 macrophages [J] . Min Pang, Guoping Zheng, Baofeng Yu, 生物化学与生物物理学报：英文版 . 2017,第005期

机译：重组CC16蛋白通过LPS激活的RAW264.7巨噬细胞中的NF-κB和p38 MAPK途径抑制促炎细胞因子的产生
4. Suppression of UHMWPE Wear Particle- induced Pro-Inflammatory Cytokine and Chemokine Production by Macrophages using an NF-κB Decoy Oligodeoxynucleotide [C] . Tzu-hua Lin, Zhenyu Yao, Sato Taishi, Society for Biomaterials annual meeting and exposition . 2014

机译：使用NF-κB诱饵寡聚脱氧核苷酸抑制UHMWPE磨损颗粒诱导的促炎性细胞因子和巨噬细胞趋化因子的产生
5. Cyclic stretch force induces periodontal ligament cells to secrete exosomes that suppress IL-1β production through the inhibition of the NF-κB signaling pathway in macrophages [D] . WANG ZHUYU 2019

机译：循环拉伸力诱导牙周膜细胞分泌外来体，通过抑制巨噬细胞中的NF-κB信号通路抑制IL-1β的产生
6. Recombinant CC16 protein inhibits the production of pro-inflammatory cytokines via NF-κB and p38 MAPK pathways in LPS-activated RAW264.7 macrophages [O] . Min Pang, Yangyang Yuan, Dong Wang, -1

机译：重组CC16蛋白通过LPS激活的RAW264.7巨噬细胞中的NF-κB和p38 MAPK途径抑制促炎细胞因子的产生
7. 2-Phenylnaphthalene Derivatives Inhibit Lipopolysaccharide-Induced Pro-Inflammatory Mediators by Downregulating of MAPK/NF-κB Pathways in RAW 264.7 Macrophage Cells. [O] . Chi-Fen Chang, Kang-Chun Liao, Chung-Hwan Chen 2017

机译：2-苯基萘衍生物通过下调RaW 264.7巨噬细胞中mapK / NF-κB途径来抑制脂多糖诱导的促炎介质。

SM905 an artemisinin derivative inhibited NO and pro-inflammatory cytokine production by suppressing MAPK and NF-κB pathways in RAW 264.7 macrophages

摘要

著录项

引文网络

相似文献

相关主题

期刊订阅