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Knockdown of CLDN6 inhibits cell proliferation and migration via PI3K/AKT/mTOR signaling pathway in endometrial carcinoma cell line HEC-1-B

机译：敲低CLDN6通过PI3K / AKT / mTOR信号通路抑制子宫内膜癌细胞株HEC-1-B中的细胞增殖和迁移

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BackgroudDysregulation of claudin-6 (CLDN6) expression in cancers has been widely documented. However, no study has reported a complete mechanistic understanding of CLDN6 regulation and function in endometrial carcinoma (EC) progression. In the current study, we aimed to assess the expression and biological functions of CLDN6 in EC.

机译：背景技术癌症中claudin-6（CLDN6）表达的失调已被广泛报道。然而，尚无研究报道对子宫内膜癌（EC）进展中CLDN6调控和功能的完整机理了解。在本研究中，我们旨在评估CLDN6在EC中的表达和生物学功能。

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期刊名称 OncoTargets and therapy
作者
Xia Cao; Guo-Zhao He;
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作者单位

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年(卷),期 2018(11),-1
年度 2018
页码 6351–6360
总页数 10
原文格式 PDF
正文语种
中图分类应用微生物学;肿瘤学;
关键词
endometrial carcinoma CLDN6 knockdown proliferation PI3K/AKT pathway prognostic invasion;

机译：子宫内膜癌;CLDN6;敲低;增殖;PI3K / AKT途径;预后;侵袭;

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专利

1. Knockdown of CLDN6 inhibits cell proliferation and migration via PI3K/AKT/mTOR signaling pathway in endometrial carcinoma cell line HEC-1-B [J] . Xia Cao, Guo-Zhao He OncoTargets and therapy . 2018,第1期

机译：敲低CLDN6通过PI3K / AKT / mTOR信号通路抑制子宫内膜癌细胞株HEC-1-B中的细胞增殖和迁移
2. Knockdown of CLDN6 inhibits cell proliferation and migration via PI3K/AKT/mTOR signaling pathway in endometrial carcinoma cell line HEC-1-B [J] . Xia Cao, Guo-Zhao He OncoTargets and therapy . 2018,第3期

机译：敲低CLDN6通过PI3K / AKT / mTOR信号通路抑制子宫内膜癌细胞株HEC-1-B中的细胞增殖和迁移
3. Downregulation of CLDN6 inhibits cell proliferation, migration, and invasion via regulating EGFR/AKT/mTOR signalling pathway in hepatocellular carcinoma [J] . Huang Lingyuan, Zhao Chanjuan, Sun Kai, Cell biochemistry and function . 2020,第5期

机译：CLDN6的下调抑制通过调节肝细胞癌中的EGFR / AKT / MTOR信号传导途径来抑制细胞增殖，迁移和侵袭
4. Label-free phosphoproteomics method to investigate signaling pathways downstream of novel PI3K/mTOR inhibitors in cancer cells [C] . Pedro Casado, Juan Carlos Rodriguez-Prados, Sabina Cosulich, American Society for Mass Spectrometry Conference on Mass Spectrometry and Allied Topics . 2011

机译：无标记的磷蛋白质方法，用于研究新型PI3K / MTOR抑制剂在癌细胞中的信号通路
5. The multi-targeted receptor tyrosine kinase inhibitor, linifanib (ABT-869), induces apoptosis and inhibits proliferation of leukemia cells through phosphoinositide-3 kinase (PI3K)/AKT-dependent signaling pathways. [D] . Hernandez, Jenny Elizabeth. 2010

机译：多靶点受体酪氨酸激酶抑制剂利尼法尼（ABT-869）通过磷酸肌醇3激酶（PI3K）/ AKT依赖性信号传导途径诱导凋亡并抑制白血病细胞的增殖。
6. CD47 Enhances Cell Viability and Migration Ability but Inhibits Apoptosis in Endometrial Carcinoma Cells via the PI3K/Akt/mTOR Signaling Pathway [O] . Yun Liu, Yue Chang, Xinhong He, 2020

机译：CD47增强了细胞活力和迁移能力但通过PI3K / AKT / MTOR信号通路抑制子宫内膜癌细胞的凋亡
7. Retracted : Knockdown of SRPX2 inhibits the proliferation, migration, and invasion of prostate cancer cells through the PI3K/Akt/mTOR signaling pathway [O] . 2018

机译：缩回：SRPX2的敲低通过PI3K / AKT / MTOR信号传导途径抑制前列腺癌细胞的增殖，迁移和侵袭

Knockdown of CLDN6 inhibits cell proliferation and migration via PI3K/AKT/mTOR signaling pathway in endometrial carcinoma cell line HEC-1-B

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