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Prespray processed cylinder inside and cylinder inside prespray processing method

机译：预喷涂处理过的气缸内部和气缸内部的预喷涂处理方法

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摘要

A cylindrical surface (3) is roughed to be prepared for a spray film (11) to be formed thereon, by cutting therein rough undulations (21) in a threaded form, using a cut chip (20) leaving a groove part (21a) of a respective undulation (21) in a preset direction or cut form, for breaking or partially tearing off a ridge part (21b) of the rough undulation (21) to have a remainder of the ridge part (21b) formed with finer undulations (22).

机译：通过在其中切割出粗糙的起伏（ 21 ），对圆柱表面（ 3 ）进行粗糙化处理，以准备在其上形成喷涂膜（ 11 ）。 >）以螺纹形式使用切屑（ 20 ）留下相应起伏（ > 21 ）以预设的方向或切割形式，用于折断或部分撕开粗糙波动（ 21> B 21 ），以使脊部的其余部分（ 21 b ）形成更细的起伏（ 22 ）。 展开▼

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公开/公告号US2003010201A1

专利类型

公开/公告日2003-01-16

原文格式PDF

申请/专利权人 TAKAHASHI HIDEO;CHUUBACHI MINORU;HASEGAWA KIYOSHI;
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申请/专利号US20020181052

发明设计人 MINORU CHUUBACHI;KIYOSHI HASEGAWA;HIDEO TAKAHASHI;
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申请日2002-07-12

分类号F16J10/00;

国家 US

入库时间 2022-08-22 00:10:05

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1. 预喷涂处理的气缸内侧和气缸内侧预喷涂处理方法 [P] . 中国专利： CN1159520C . 2004.07.28

2. 预喷涂处理的气缸内侧和气缸内侧预喷涂处理方法 [P] . 中国专利： CN1395652A . 2003-02-05

3. PRESPRAY PROCESSED CYLINDER INSIDE AND CYLINDER INSIDE PRESPRAY PROCESSING METHOD [P] . 韩国专利： KR100624541B1 . 2006-09-18

机译：预喷雾内筒及内喷雾筒预喷雾方法

4. PRESPRAY PROCESSED CYLINDER INSIDE AND CYLINDER INSIDE PRESPRAY PROCESSING METHOD [P] . 欧洲知识产权局专利： EP1334268A1 . 2003-08-13

机译：预喷雾内筒及内喷雾筒预喷雾方法

5. Prespray processed cylinder inside and cylinder inside prespray processing method [P] . 美国专利： US6622685B2 . 2003-09-23

机译：预喷涂处理过的气缸内部和气缸内部的预喷涂处理方法

1. Methodologies for the Evaluation of Systematic Form Deviations for Inside-Diameter Cylindrical Features and Their Relationship to Process Variables [C] . R.P.Henke, R.M.Cassou, K.D.Summerhays, Annual meeting on American Society for Precision Engineering . 1996

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3. Role of Rotating Cylinder toward Mixed Convection inside a Wavy Heated Cavity via Two-Phase Nanofluid Concept [O] . Ammar I. Alsabery, Mohammad Ghalambaz, Taher Armaghani, 2020

4. Experimental Investigation of the In-Cylinder Tumble Motion inside GDI Cylinder at Different Planes under Steady-State Condition using Stereoscopic-PIV [O] . Mohammed El Adawy, M. R. Heikal, A. Rashid A. Aziz 2019

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1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004）（P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期

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5. Analysis and Optimization of Inside-Cushion Structure in High-Speed Hydraulic Cylinders [J] . Ruqi Ding ,Sheng Wang ,Ying Wei . 北京理工大学学报：英文版 . 2019,第4期

6. 通过内部气流及气缸壁面温度测量与分析提高冰箱压缩机性能 [C] . Cheng Rui ,程锐 ,Xiao Rui . 2015年中国家用电器技术大会 . 2015

7. 机车气缸套等离子喷涂再制造涂层的制备及摩擦磨损性能的研究 [A] . 徐海峰 . 2017