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Kit for whitening a body surface of a user, related method and process

机译：用于美白使用者身体表面的套件，相关方法和过程

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摘要

Kit for whitening a body surface of a user, comprising:an oxidizing composition application device (12);wherein the kit comprises an activator sheet (14) which is movable independently of the oxidizing composition application device (12), the activator sheet (14) comprising an outer deformable substrate (50) intended to be applied on an oxidizing composition (34) placed on the skin of the user, the activator sheet (14) comprising an activator composition (52) carried by the outer substrate (50).

机译：用于增白使用者身体表面的套件，包括：氧化剂组合物施用装置（ 12 ）; 其中试剂盒包含活化剂片（< B> 14 ）可以独立于氧化剂组合物施加装置（ 12 ）移动，活化剂片（ 14 ）包括外部可变形基材（ 50 ）用于施加在放置在使用者皮肤上的氧化性组合物（ 34 ）上，活化剂片（ 14 ）包含活化剂组合物（ 52 ）由外部基板（ 50 ）承载。 展开▼

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公开/公告号US10016345B2

专利类型

公开/公告日2018-07-10

原文格式PDF

申请/专利权人 LOREAL;
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申请/专利号US201414906676

发明设计人 PRASUN BANDYOPADHYAY;GAURAV AGARWAL;SHOIBAL PATTANAIK;CAROLINE DELAUNAY;
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申请日2014-07-15

分类号A61K8/02;A61K8/22;A45D44/00;A61Q19/02;A61K8/23;

国家 US

入库时间 2022-08-21 13:04:13

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1. 用于美白使用者身体表面的成套产品、相关方法和过程 [P] . 中国专利： CN105407975A . 2016-03-16

2. 牙齿美白设备、用于所述牙齿美白设备的可佩戴套件以及用于所述牙齿美白设备的平台 [P] . 中国专利： CN101563046A . 2009-10-21

3. Kit for whitening the body surface of a user, related methods and processes [P] . 日本专利： JP2016525525A . 2016-08-25

机译：美白使用者身体表面的套件，相关方法和过程

4. KIT FOR WHITENING A BODY SURFACE OF A USER, RELATED METHOD AND PROCESS [P] . 韩国专利： KR20160033113A . 2016-03-25

机译：用于美化用户身体表面的套件，相关方法和过程

5. KIT FOR WHITENING A BODY SURFACE OF A USER RELATED METHOD AND PROCESS [P] . IN201617001396A . 2016-07-29

机译：用于美化使用者身体表面的套件相关的方法和过程

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4. Comparison of the TEMPO+ ear-level speech processor and the cis pro+ body-worn processor in adult MED-EL cochlear implant users. [J] . Helms J, Muller J, Schon F, ORL: Journal for oto-rhino-laryngology and its borderlands . 2001,第1期

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1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004）（P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期

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