首页> 外文OA文献 >IDH2 compensates for IDH1 mutation to maintain cell survival under hypoxic conditions in IDH1‑mutant tumor cells

【2h】

IDH2 compensates for IDH1 mutation to maintain cell survival under hypoxic conditions in IDH1‑mutant tumor cells

机译：IDH2补偿IDH1突变，以在IDH1-突变肿瘤细胞中的缺氧条件下维持细胞存活

代理获取

本网站仅为用户提供外文OA文献查询和代理获取服务，本网站没有原文。下单后我们将采用程序或人工为您竭诚获取高质量的原文，但由于OA文献来源多样且变更频繁，仍可能出现获取不到、文献不完整或与标题不符等情况，如果获取不到我们将提供退款服务。请知悉。

页面导航

著录项
引文网络
相似文献
相关主题

著录项

作者
Yao Zhang; Weifeng Lv; Qi Li; Qinhao Wang; Yi Ru; Xin Xiong; Fengqi Yan; Tao Pan; Wei Lin; Xia Li;
展开▼
作者单位

展开▼
年度 2019
总页数
原文格式 PDF
正文语种
中图分类

相似文献

外文文献
中文文献
专利

1. IDH2 compensates for IDH1 mutation to maintain cell survival under hypoxic conditions in IDH1-mutant tumor cells [J] . Zhang Yao, Lv Weifeng, Li Qi, Nature reviews neuroscience . 2019,第2期

机译：IDH2补偿IDH1突变，以在IDH1-突变肿瘤细胞中的缺氧条件下保持细胞存活
2. Pyrosequencing of IDH1 and IDH2 Mutations in Brain Tumors and Non-neoplastic Conditions [J] . Matthew D. Cykowski, Richard A. Allen, Kar-Ming Fung, Diagnostic molecular pathology : . 2012,第4期

机译：脑肿瘤和非肿瘤性疾病中IDH1和IDH2突变的焦磷酸测序
3. Primary acute myeloid leukemia cells with IDH1 or IDH2 mutations respond to a DOT1L inhibitor in vitro [J] . S M Sarkaria, M J Christopher, J M Klco, Leukemia . 2014,第12期

机译：IDH1 或 IDH2 突变的原发性急性髓性白血病细胞在体外对DOT1L抑制剂有反应
4. Identification of interaction networks of mutant and wild-type IDH1 in glioma cell lines [C] . Nina Overbeck, Anja Stefanski, Vanessa Scherbaum, ASMS Conference on Mass Spectrometry and Allied Topics . 2016

机译：胶质瘤细胞系中突变体和野生型IDH1相互作用网络的鉴定
5. PTHR1/SOX9? and IDH1/IDH2 Relative Expression in Primary Chondrocyte and Chondrosarcoma Cells Under the Synergistic Influence of Inducible Hypoxia and Extracellular Acidosis [D] . Vangjeli, Kostika. 2020

机译：pthr1 / sox9？诱导缺氧和细胞外酸中毒的协同影响下，IDH1 / IDH2在原发性软骨细胞和软骨肉瘤细胞中的相对表达
6. Mutational Hotspot of TET2 IDH1 IDH2 SRSF2 SF3B1 KRAS and NRAS from Human Systemic Mastocytosis Are Not Conserved in Canine Mast Cell Tumors [O] . Eleonora Zorzan, Katia Hanssens, Mery Giantin, -1

机译：犬肥大细胞瘤中TET2IDH1IDH2SRSF2SF3B1KRAS和NRAS来自人类系统性细胞增生作用的突变热点不保守。
7. Mutational Hotspot of TET2, IDH1, IDH2, SRSF2, SF3B1, KRAS, and NRAS from Human Systemic Mastocytosis Are Not Conserved in Canine Mast Cell Tumors [O] . Zorzan, Eleonora, Hanssens, Katia, Giantin, Mery, 2015

机译：犬肥大细胞瘤中TET2，IDH1，IDH2，SRSF2，SF3B1，KRAS和NRAS来自人类系统性细胞增生作用的突变热点不保守。

IDH2 compensates for IDH1 mutation to maintain cell survival under hypoxic conditions in IDH1‑mutant tumor cells

著录项

引文网络

相似文献

相关主题

期刊订阅