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Lack of cross-desensitization between leptin and prolactin signaling pathways despite the induction of suppressor of cytokine signaling 3 and PTP-1B

机译：尽管细胞因子信号传导3和PTP-1B的抑制剂诱导抑制抑制剂，但瘦蛋白和催乳素信号传导途径之间缺乏杂交。

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A F Roy; Y Benomar; V Bailleux; C M Vacher; A Aubourg; A Gertler; J Djiane; M Taouis;
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机译：细胞因子信号1 / JAB的抑制剂和细胞因子信号3 /细胞因子诱导的SH2含蛋白3的抑制剂负面调节正常人表皮角蛋白的信号转导和转录信号通路的激活剂。
2. Activation of STAT signaling pathways and induction of suppressors of cytokine signaling (SOCS) proteins in mammalian lens by growth factors. [J] . Ebong S, Yu CR, Carper DA, Investigative ophthalmology & visual science . 2004,第3期

机译：生长因子在哺乳动物晶状体中激活STAT信号通路并诱导细胞因子信号（SOCS）蛋白抑制剂的产生。
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机译：细胞因子信号转导抑制剂7通过与STAT5或STAT3相互作用并减弱其核易位来抑制催乳激素，生长激素和瘦素信号转导。

Lack of cross-desensitization between leptin and prolactin signaling pathways despite the induction of suppressor of cytokine signaling 3 and PTP-1B

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