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Acute myocardial injury from carbon monoxide poisoning by cardiac magnetic resonance imaging

机译:心脏磁共振成像对一氧化碳中毒的急性心肌损伤

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摘要

A 40-year-old patient presented to our emergency department with altered mental status and supraventricular tachycardia. His carboxyhaemoglobin level was detected at 14%. He was found to have developed myocardial injury with a typical troponin rise and fall peaking at 22.6 ng/mL. Coronary angiography demonstrated normal coronary arteries. A battery of inflammatory and viral serologies was also negative. Cardiac magnetic resonance (CMR) was performed to evaluate the patient's left ventricular function and myocardial damage. Steady-state free-precision cine demonstrated a mildly depressed left ventricular systolic function (ejection fraction of 54%) with hypokinesis of the anterior wall and regional akinesis of the.inferior wall (Supplementary data online, Videos S1 and S2). First-pass perfusion demonstrated even perfusion throughout all wall segments (Supplementary data online, Video S3). Late gadolinium-enhancement images demonstrated multiple focal areas of high signal consistent with myocardial necrosis or fibrosis (Panels A-D).
机译:一名40岁的患者因精神状态改变和室上性心动过速而出现在我们的急诊科。他的羧基血红蛋白水平为14%。发现他发展为心肌损伤,典型的肌钙蛋白上升和下降峰值为22.6 ng / mL。冠状动脉造影显示冠状动脉正常。一系列炎性和病毒血清学也为阴性。进行心脏磁共振(CMR)以评估患者的左心室功能和心肌损害。稳态自由电影显示左室收缩功能轻度下降(射血分数为54%),前壁运动减退和下壁区域运动(在线补充数据,视频S1和S2)。首次通过灌注显示了所有壁段的均匀灌注(在线补充数据,视频S3)。晚期g增强图像显示出高信号的多个病灶区域,与心肌坏死或纤维化一致(图A-D)。

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