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机译:干扰HSF1 / HSP70 / BAG3途径将胶质瘤细胞引发至基质脱离和BH3仿真诱导的细胞凋亡
Goethe Univ Hosp Expt Neurosurg Frankfurt Germany;
Goethe Univ Hosp Expt Neurosurg Frankfurt Germany;
Goethe Univ Hosp Radiotherapy &
Oncol Frankfurt Germany;
Goethe Univ Hosp Dr Senckenberg Inst Neurooncol Frankfurt Germany;
Goethe Univ Hosp Dr Senckenberg Inst Neurooncol Frankfurt Germany;
Goethe Univ Hosp Inst Expt Canc Res Pediat Frankfurt Germany;
Goethe Univ Hosp Dr Senckenberg Inst Neurooncol Frankfurt Germany;
Goethe Univ Hosp Dept Neurosurg Frankfurt Germany;
Goethe Univ Hosp Radiotherapy &
Oncol Frankfurt Germany;
German Canc Consortium DKTK Heidelberg Germany;
Goethe Univ Hosp Expt Neurosurg Frankfurt Germany;
机译:干扰HSF1 / HSP70 / BAG3途径将胶质瘤细胞引发至基质脱离和BH3仿真诱导的细胞凋亡
机译:西仑吉肽通过抑制FAK / src / AKT途径介导内皮细胞和神经胶质瘤细胞的细胞脱离和凋亡
机译:膳食黄酮非瑟酮通过阻断HSF1与hsp70启动子的结合来抑制HSF1活性,从而诱导癌细胞凋亡。
机译:Ugonin K通过在人体皮肤癌细胞中的活性氧物质/凋亡信号途径诱导细胞周期停滞和细胞凋亡
机译:蛋白激酶Chk2是由上皮细胞脱离细胞外基质引发的肠道上皮细胞死亡的介体
机译:BAG3的下调通过BAG3 / HSP70 /HIF-1α信号通路抑制增殖并促进胶质母细胞瘤的凋亡
机译:干扰HSF1 / HSP70 / BAG3途径将胶质瘤细胞引发至基质脱离和BH3仿真诱导的细胞凋亡