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机译:通过TLR4 / MyD88 / NF-Kappa B和MAPK / Caspase信号传导途径防止炎症,氧化应激和凋亡,Lealiritin通过预防炎症,氧化应激和凋亡来抑制UVB诱导的皮肤损伤
Nanjing Med Univ Dept Dermatol Affiliated Huaian Peoples Hosp 1 6 Beijing Rd West Huaian 223300;
Nanjing Med Univ Dept Dermatol Affiliated Huaian Peoples Hosp 1 6 Beijing Rd West Huaian 223300;
Nanjing Med Univ Dept Dermatol Affiliated Huaian Peoples Hosp 1 6 Beijing Rd West Huaian 223300;
Nanjing Med Univ Dept Dermatol Affiliated Huaian Peoples Hosp 1 6 Beijing Rd West Huaian 223300;
Nanjing Med Univ Dept Dermatol Affiliated Huaian Peoples Hosp 1 6 Beijing Rd West Huaian 223300;
Nanjing Med Univ Dept Dermatol Affiliated Huaian Peoples Hosp 1 6 Beijing Rd West Huaian 223300;
Nanjing Med Univ Dept Dermatol Affiliated Huaian Peoples Hosp 1 6 Beijing Rd West Huaian 223300;
ultraviolet B radiation; liquiritin; inflammation; oxidative stress; apoptosis;
机译:通过TLR4 / MyD88 / NF-Kappa B和MAPK / Caspase信号传导途径防止炎症,氧化应激和凋亡,Lealiritin通过预防炎症,氧化应激和凋亡来抑制UVB诱导的皮肤损伤
机译:通过通过TLR4 / MyD88 / TRIF / NLRP3信号通路抑制炎症和氧化胁迫,从脊柱孔Gleditsiae中分离出的5 o-methyldihydroquetin和Cilicicone B.从脊柱杆菌和脂多糖诱导小鼠急性肾损伤
机译:通过通过TLR4 / MyD88 / TRIF / NLRP3信号通路抑制炎症和氧化胁迫,从脊柱孔Gleditsiae中分离出的5 o-methyldihydroquetin和Cilicicone B.从脊柱杆菌和脂多糖诱导小鼠急性肾损伤
机译:在人肺和皮肤成纤维细胞中研究了炎症和氧化应激信号转导中的MAPK ERK,JNK和P38。
机译:Liquiritin可通过TLR4 / MyD88 /NF-κB和MAPK / caspase信号通路阻止炎症氧化应激和凋亡从而抑制UVB诱导的皮肤损伤
机译:通过TLR4 / MYD88 / NF-κB和MAPK / Caspase信号传导途径防止炎症,氧化应激和凋亡,Lealiritin通过预防炎症,氧化应激和凋亡抑制UVB诱导的皮肤损伤