机译:L L OS FFAR FFAR 2通过炎症抑制剂的表观遗传失调促进结肠癌
Division of Hematology and Oncology Department of MedicineMedical College of WisconsinMilwaukee WI;
Department of PathologyJohn Hopkins UniversityBaltimore MD;
Department of Obstetrics and GynecologyMedical College of WisconsinMilwaukee WI;
Division of Hematology and Oncology Department of MedicineMedical College of WisconsinMilwaukee WI;
Division of Hematology and Oncology Department of MedicineMedical College of WisconsinMilwaukee WI;
Division of Hematology and Oncology Department of MedicineMedical College of WisconsinMilwaukee WI;
Center for BiostatisticsThe Ohio State UniversityColumbus OH;
Department of PathologyOhio State UniversityColumbus OH;
Division of Hematology Department of Internal Medicine College of MedicineComprehensive Cancer;
Division of Hematology and Oncology Department of MedicineMedical College of Wisconsin RM C4930;
FFAR2; colon cancer; epigenetics; HDAC; butyrate;
机译:L L OS FFAR FFAR 2通过炎症抑制剂的表观遗传失调促进结肠癌
机译:改善或恶化:癌症和糖尿病中的FFAR1和FFAR4信号传导
机译:无论好坏:FFAR1和FFAR4在癌症和糖尿病中的信号传导
机译:表观遗传开关涉及由Myc和miRNA-17-92 microRNA簇影响的将炎症与癌症联系起来的正反馈回路
机译:免疫失调和胰腺癌:免疫功能过度,炎症和表观遗传修饰
机译:FFAR2的丢失通过炎症抑制因子的表观遗传失调促进结肠癌
机译:维生素C促进去离菜或氮杂扎氨酸诱导的DNA羟甲基甲基化,随后的结肠癌细胞中的外延沉默的肿瘤抑制剂CDKN1a的再活化