机译:直肠癌中GPRC5a表达的升高通过VNN-1诱导氧化应激促进肿瘤进展
East China Normal Univ 500 Dongchuan Rd Shanghai 200241 Peoples R China;
East China Normal Univ 500 Dongchuan Rd Shanghai 200241 Peoples R China;
East China Normal Univ 500 Dongchuan Rd Shanghai 200241 Peoples R China;
East China Normal Univ 500 Dongchuan Rd Shanghai 200241 Peoples R China;
East China Normal Univ 500 Dongchuan Rd Shanghai 200241 Peoples R China;
Tongji Univ Sch Med Dept Gastroenterol Shanghai East Hosp Shanghai 200120 Peoples R China;
East China Normal Univ 500 Dongchuan Rd Shanghai 200241 Peoples R China;
East China Normal Univ 500 Dongchuan Rd Shanghai 200241 Peoples R China;
Shanghai Jiao Tong Univ Key Lab Cell Differentiat &
Apoptosis Chinese Minist Educ Dept;
East China Normal Univ 500 Dongchuan Rd Shanghai 200241 Peoples R China;
Southern Med Univ Fengxian Hosp Shanghai 201499 Peoples R China;
East China Normal Univ 500 Dongchuan Rd Shanghai 200241 Peoples R China;
East China Normal Univ 500 Dongchuan Rd Shanghai 200241 Peoples R China;
East China Normal Univ 500 Dongchuan Rd Shanghai 200241 Peoples R China;
GPRC5A; VNN1; oxidative stress; NF-B; colorectal cancer;
机译:直肠癌中GPRC5a表达的升高通过VNN-1诱导氧化应激促进肿瘤进展
机译:通过调节Wnt /β-catenin信号传导途径,高PLK4表达促进肿瘤进展并诱导上皮-间质转化
机译:miR-137抑制诱导的Paxillin过表达促进结直肠癌的肿瘤进展和转移
机译:超过750个基因的高深度测序支持肝脏有限转移性结直肠癌大多数患者的原发性肿瘤和转移的线性进展
机译:H3K18和H4K12的乙酰化以及HDAC2的表达和结直肠癌的进展。
机译:肿瘤诱导的髓样来源的抑制细胞通过人类大肠癌的氧化代谢促进肿瘤进展
机译:结直肠癌中GPRC5a表达的升高通过VNN-1诱导氧化应激促进肿瘤进展