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机译:CDKL5缺乏通过放松SMAD3信号传导使神经元预测细胞死亡
Department of Biomedical and Neuromotor SciencesUniversity of BolognaBologna Italy;
Department of Biomedical and Neuromotor SciencesUniversity of BolognaBologna Italy;
Department of Biomedical and Neuromotor SciencesUniversity of BolognaBologna Italy;
Department of Biomedical and Neuromotor SciencesUniversity of BolognaBologna Italy;
Department of Biomedical and Neuromotor SciencesUniversity of BolognaBologna Italy;
Department of Biomedical and Neuromotor SciencesUniversity of BolognaBologna Italy;
Department of Biomedical and Neuromotor SciencesUniversity of BolognaBologna Italy;
Department of Biomedical and Neuromotor SciencesUniversity of BolognaBologna Italy;
Department of Biomedical and Neuromotor SciencesUniversity of BolognaBologna Italy;
CDKL5; hippocampal neurons; neuronal survival; neuronal maturation; SMAD3; TGF‐β signaling;
机译:CDKL5缺乏通过放松SMAD3信号传导使神经元预测细胞死亡
机译:SBDS缺乏导致活性氧的失调,导致细胞死亡增加和细胞生长减少。
机译:TrkB信号传导是中枢神经系统神经元出生后生存所必需的,并保护海马和运动神经元免受轴切术诱导的细胞死亡。
机译:烟草植物中的牺牲细胞死亡和内源性二次细胞死亡信号的分离
机译:信号转导受体Gp130对于保护视网膜神经元免于应激诱导的细胞死亡至关重要,但对于视网膜发育却至关重要。
机译:TrkB信号对于中枢神经系统神经元的产后生存是必需的并保护海马和运动神经元免受轴突切开术诱导的细胞死亡。
机译:细胞外信号调节激酶的组成性激活使弥漫性大B细胞淋巴瘤细胞系易于CD40介导的细胞死亡。