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Congenital programs of the behavior and nontrivial quantum effects in the neurons work

机译:神经元工作中行为和非平凡量子效应的先天性程序

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The problem of processing and transmitting information within neurons is considered. The fundamental paradox of molecular biology is formulated, namely that interactions between biologically important molecules should lead to an exponentially large number of variants of molecular structures, but only a small number of them are biologically relevant. The problem is that the known interaction potentials between atoms do not allow this. The solution of the paradox, based on the nonlinear quantum model of interaction between biologically important molecules, is proposed. The model includes a nonlinear equation for many-particle nonlocal potential describing this interaction. Under the action of this potential there occurs the formation of synaptic connections between neurons, and transport processes and molecular recognition inside neurons. The information on which programs of innate behavior operate is hypothesized to reside in the quantum degrees of freedom of the proposed potential. Possible experiments to test the model are proposed.
机译:考虑了在神经元内处理和传输信息的问题。提出了分子生物学的基本悖论,即生物学重要分子之间的相互作用应导致分子结构的大量变体,但其中只有少数是生物学相关的。问题在于原子之间已知的相互作用势不允许这样做。提出了基于生物学重要分子之间相互作用的非线性量子模型的悖论解决方案。该模型包括一个描述该相互作用的多粒子非局部电势的非线性方程。在这种潜力的作用下,神经元之间会发生突触连接,并在神经元内部发生转运过程和分子识别。假定先天行为程序在其上起作用的信息驻留在所提议电位的量子自由度中。提出了测试模型的可能实验。

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