Ginsenoside Rg1-induced activation of astrocytes promotes functional recovery via the PI3K/Akt signaling pathway following spinal cord injury

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Ginsenoside Rg1-induced activation of astrocytes promotes functional recovery via the PI3K/Akt signaling pathway following spinal cord injury

机译：人参皂甙RG1诱导的星形胶质细胞的活化通过PI3K / AKT信号通路在脊髓损伤后通过PI3K / AKT信号通路促进功能性回收

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Aims: To determine whether ginsenoside Rg1 is involved in scratch wound healing through altered expression of related molecules in astrocytes and improved functional recovery after spinal cord injury (SCI).

机译：目的：通过改变星形胶质细胞中的相关分子的表达和脊髓损伤后的功能恢复改善脊髓损伤（SCI）的功能恢复，确定人参皂苷RG1是否参与划痕伤口愈合。

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《Life sciences》 |2020年第2020期|共9页
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正文语种 eng
中图分类医药、卫生;
关键词
Ginsenoside Rg1; Astrocytes; Neurotrophic factor; Spinal cord injury; Repair;

机译：人参皂苷Rg1;星形胶质细胞;神经营养因子;脊髓损伤;修理;

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1. Ginsenoside Rg1-induced activation of astrocytes promotes functional recovery via the PI3K/Akt signaling pathway following spinal cord injury [J] . Life sciences . 2020,第期

机译：人参皂甙RG1诱导的星形胶质细胞的活化通过PI3K / AKT信号通路在脊髓损伤后通过PI3K / AKT信号通路促进功能性回收
2. Neuron and microglia/macrophage-derived FGF10 activate neuronal FGFR2/PI3K/Akt signaling and inhibit microglia/macrophages TLR4/NF-κB-dependent neuroinflammation to improve functional recovery after spinal cord injury [J] . Jian Chen, Zhouguang Wang, ZengMing Zheng, Cell death & disease. . 2017,第10期

机译：神经元和微胶质细胞/巨噬细胞衍生的FGF10激活神经元FGFR2 / PI3K / AKT信号传导和抑制小胶质细胞/巨噬细胞TLR4 / NF- κ-/ i> B依赖性神经炎性，以改善脊髓损伤后的功能恢复
3. Lentivirus-Mediated Overexpression of miR-29a Promotes Axonal Regeneration and Functional Recovery in Experimental Spinal Cord Injury via PI3K/Akt/mTOR Pathway [J] . Yin Hua, Shen Liming, Xu Chao, Neurochemical research . 2018,第11期

机译：慢病毒介导的miR-29a过表达促进了通过PI3K / AKT / MTOR途径在实验脊髓损伤中的轴突再生和功能性回收
4. Determining the functional role of keratin filaments in apoptosis via the PI3K/Akt signaling pathway using LTQ Orbitrap MS/MS analysis [C] . Nancy Fernandes, Nicole Morin Jaskiewicz, David H. Townson, ASMS Conference on Mass Spectrometry and Allied Topics . 2014

机译：使用LTQ orbitrap MS / MS分析确定通过PI3K / AKT信号通路凋亡中角蛋白长丝在细胞凋亡中的功能作用
5. Transduced fibroblasts over-expressing MMP-3 increase axonal fibers traversing the injury site and promote functional recovery after spinal cord injury. [D] . Pizzi, Michael Anthony. 2006

机译：过度表达MMP-3的转导成纤维细胞增加了穿越损伤部位的轴突纤维并促进了脊髓损伤后的功能恢复。
6. Neuron and microglia/macrophage-derived FGF10 activate neuronal FGFR2/PI3K/Akt signaling and inhibit microglia/macrophages TLR4/NF-κB-dependent neuroinflammation to improve functional recovery after spinal cord injury [O] . Jian Chen, Zhouguang Wang, ZengMing Zheng, 2017

机译：神经元和小胶质细胞/巨噬细胞衍生的FGF10激活神经元FGFR2 / PI3K / Akt信号传导并抑制小胶质细胞/巨噬细胞TLR4 /NF-κB依赖性神经炎症以改善脊髓损伤后的功能恢复
7. Methylprednisolone promotes recovery of neurological function after spinal cord injury: association with Wnt/β-catenin signaling pathway activation [O] . Gong-biao Lu, Fu-wen Niu, Ying-chun Zhang, 2016

机译：甲基强的松龙促进脊髓损伤后神经功能的恢复：与Wnt /β-catenin信号通路激活的关联

Ginsenoside Rg1-induced activation of astrocytes promotes functional recovery via the PI3K/Akt signaling pathway following spinal cord injury

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