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首页> 外文期刊>Journal of cardiovascular electrophysiology >Iterative atrial tachycardia originating from the coronary sinus musculature.
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Iterative atrial tachycardia originating from the coronary sinus musculature.

机译:源自冠状窦肌肉组织的反复性房性心动过速。

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摘要

A case of iterative atrial tachycardia leading to dilated cardiomyopathy is reported. During electrophysiologic study, the tachycardia showed a markedly irregular cycle length associated with changes in atrial activation breakthrough as demonstrated by coronary sinus (CS) recordings and frequently degenerated into self-terminating atrial fibrillation. Left atrial transseptal mapping demonstrated the earliest endocardial atrial activation close to the posterolateral mitral annulus, but this was invariably later than that recorded within the CS, where low-energy radiofrequency applications eliminated the tachycardia. No acute vessel damage was observed at postablation CS angiography. In accordance with previously published experimental data, we hypothesized that the muscular sleeves surrounding the CS might be involved in the genesis of this tachycardia. During 6-month follow-up, the patient remained asymptomatic without tachycardia recurrences and with complete recovery of left ventricular function, confirming the reversible nature of the tachycardia-induced cardiomyopathy.
机译:据报道一例导致房性心肌病的反复心房性心动过速。在电生理研究过程中,心动过速显示出明显不规则的周期长度,与冠状窦(CS)记录所证明的心房激活突破性变化有关,并且经常退化为自我终止的心房颤动。左心房前房间隔图显示最早的心内膜心房激活接近二尖瓣后外侧环,但这总是比CS中记录的要晚,在CS中,低能量射频应用消除了心动过速。消融后CS血管造影未观察到急性血管损伤。根据先前发布的实验数据,我们假设CS周围的肌肉袖可能参与了这种心动过速的发生。在6个月的随访中,患者无症状,无心动过速复发,左心室功能完全恢复,证实了心动过速诱发的心肌病的可逆性。

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