首页> 美国卫生研究院文献>Journal of Cardiology Cases >Interesting electrophysiological findings in a patient with coexistence of atrial tachycardia originating from coronary sinus and slow-fast atrioventricular nodal reentrant tachycardia
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Interesting electrophysiological findings in a patient with coexistence of atrial tachycardia originating from coronary sinus and slow-fast atrioventricular nodal reentrant tachycardia

机译:由冠状窦和慢速房室结折返性心动过速并发的房性心动过速并存的患者中有趣的电生理发现

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摘要

Slow-fast atrioventricular nodal tachycardia (AVNRT) has various electrophysiological aspects due to atrioventricular (AV) nodal physiology. In addition, concomitantly another form of arrhythmia with AVNRT, especially atrial tachycardia (AT), was an infrequent arrhythmia. A 38-year-old female with narrow QRS tachycardia underwent electrophysiological study due to frequent faintness. The electrophysiological study disclosed the coexistence of AT originating from coronary sinus (CS) with slow-fast AVNRT. We easily diagnosed AT originating from CS and terminated with several radiofrequency ablations (RFA) around CS. The diagnosis of slow-fast AVNRT, however, was somewhat difficult due to the following findings: (1) small amount of adenosine triphosphate (ATP) could terminate slow-fast AVNRT reproducibly; (2) we could provoke slow-fast AVNRT only by RV pacing with isoproterenol infusion. With other electrophysiological findings, we diagnosed slow-fast AVNRT. Radiofrequency energy was delivered initially in the posteroseptal region, followed by inside CS, and finally in the middle septal region, which completed the slow pathway ablation. After the procedure, we could never provoke these arrhythmias.<>Learning objective: Coexistence of focal AT originating from CS with slow-fast AVNRT is a rare phenomenon. Furthermore, slow-fast AVNRT could show unusual characteristic as following: (1) small amount of ATP terminates slow-fast AVNRT; (2) atrial pacing never provoked slow-fast AVNRT with isoproterenol infusion whereas ventricular pacing did, which depends on the physiological characteristic of the dual AV nodal pathway. Accordingly, we should precisely assess the obtained electrophysiological findings.>
机译:慢速房室结性心动过速(AVNRT)由于房室结(AV)节点生理而具有各种电生理学方面。此外,伴有AVNRT的另一种心律不齐,尤其是房性心动过速(AT),是一种罕见的心律失常。一名38岁的QRS狭窄心动过速的女性因频繁晕厥而接受了电生理研究。电生理研究揭示了源自冠状窦(CS)的AT与慢速AVNRT并存。我们很容易诊断出源自CS的AT,并终止于CS周围的多个射频消融(RFA)。然而,由于以下发现,对慢速AVNRT的诊断有些困难:(1)少量的三磷酸腺苷(ATP)可以可再现地终止慢速AVNRT; (2)我们只能通过右心室起搏和异丙肾上腺素输注来诱发慢速AVNRT。结合其他电生理发现,我们诊断出慢速AVNRT。射频能量最初在后中隔区域内传递,然后在内部CS内传递,最后在中隔区域传递,这完成了慢路径消融。手术后,我们再也不会激起这些心律失常。 strong>学习目标:源自CS的局灶性AT与慢速AVNRT并存是一种罕见的现象。此外,慢速AVNRT可能表现出以下异常特征:(1)少量的ATP终止了慢速AVNRT; (2)心房起搏从未引起异丙肾上腺素输注的慢速AVNRT,而心室起搏却引起了,这取决于双AV淋巴结通路的生理特征。因此,我们应该精确评估获得的电生理结果。

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