Possible mechanisms explaining the tendency towards interstitial fibrosis in aristolochic acid-induced acute tubular necrosis

Li Yang Xiaomei Li and Haiyan Wang

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Possible mechanisms explaining the tendency towards interstitial fibrosis in aristolochic acid-induced acute tubular necrosis

机译：解释马兜铃酸诱导的急性肾小管坏死间质纤维化趋势的可能机制

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Background. We explored possible mechanisms responsible for the inability of plerosis and the tendency towards fibrosis in aristolochic acid-induced acute tubular necrosis (AA-ATN).

机译：背景。我们探索了可能的机制，导致马兜铃酸诱导的急性肾小管坏死（AA-ATN）的无力致病和纤维化趋势。

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《Nephrology Dialysis Transplantation》 |2007年第2期|445-456|共12页
作者
Li Yang Xiaomei Li and Haiyan Wang;
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Renal Division Department of Medicine Peking University First Hospital Beijing PR China;

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专利

1. Possible mechanisms explaining the tendency towards interstitial fibrosis in aristolochic acid-induced acute tubular necrosis. [J] . Yang L, Li X, Wang H Nephrology, dialysis, transplantation: official publication of the European Dialysis and Transplant Association - European Renal Association . 2007,第2期

机译：可能的机制解释了马兜铃酸诱导的急性肾小管坏死的间质纤维化趋势。
2. Gene Expression in Biopsies of Acute Rejection and Interstitial Fibrosis/Tubular Atrophy Reveals Highly Shared Mechanisms That Correlate With Worse Long-Term Outcomes [J] . Modena B. D., Kurian S. M., Gaber L. W., American journal of transplantation: official journal of the American Society of Transplantation and the American Society of Transplant Surgeons . 2016,第7期

机译：急性排斥反应和间质纤维化/小管萎缩的活检中的基因表达揭示了与较差的长期结果相关的高度共享的机制
3. CONCOMITANT ACUTE TUBULAR INTERSTITIAL NEPHRITIS AND ACUTE TUBULAR NECROSIS IN A CHILD TREATED WITH VANCOMYCIN AND PIPERACILLIN/TAZOBACTAM [J] . Varier I., Mertens C., Steele R., Journal of investigative medicine . 2018,第2期

机译：伴随着Vancomycin和Piperacillin / Tazobactam治疗儿童急性管状间质性肾炎和急性管状坏死
4. Prediction of renal transplant rejection and acute tubular necrosis in renal transplant based on SVM [C] . Li Xun, Wang Yao, Wang Chengxuan, BMEI 2012;International Conference on Biomedical Engineering and Informatics . 2012

机译：基于支持向量机的肾脏移植排斥反应和急性肾小管坏死的预测
5. Mechanisms of targeted necrosis of proximal straight tubular cells following ischemic renal injury. [D] . Devalaraja-Narashimha, Kishor. 2008

机译：缺血性肾损伤后近端直小管细胞靶向坏死的机制。
6. Gene Expression in Biopsies of Acute Rejection and Interstitial Fibrosis/Tubular Atrophy Reveals Highly Shared Mechanisms That Correlate With Worse Long-Term Outcomes [O] . B. D. Modena, S. M. Kurian, L. W. Gaber, -1

机译：急性排斥反应和间质纤维化/小管萎缩的活检中的基因表达揭示了与较差的长期结果相关的高度共享的机制
7. Possible mechanisms explaining the tendency towards interstitial fibrosis in aristolochic acid-induced acute tubular necrosis [O] . L. Yang, X. Li, H. Wang 2006

机译：可能的机制，解释了促型酸诱导的急性管状坏死中的间质纤维化倾向

Possible mechanisms explaining the tendency towards interstitial fibrosis in aristolochic acid-induced acute tubular necrosis

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