The effect of cytoplasmic regions of LIF receptor on activating MAPK p42/44 in HL-60 cell

LIU Hou-qi; SHAN Ji-kuan; TANG Shu-ping

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The effect of cytoplasmic regions of LIF receptor on activating MAPK p42/44 in HL-60 cell

机译：LIF受体胞质区对HL-60细胞激活MAPK p42 / 44的影响

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Objective: To study the relation between the cytoplasmic region of each subunit of leukemia inhibitory factor (LIF) receptor and mitogen-activated protein kinase (MAPK) in human leukemia line HL-60 for studying the mechnaism of leukemia cell proliferation disorder. Methods: We prepared the chimerical receptor with exchanged cytoplasimc do- main (190/130, 130/190), expressed it on the membrane of HL-60, and then bound LIF with the wild type receptor compet- itively.

机译：目的：研究人白血病细胞系HL-60中白血病抑制因子（LIF）受体每个亚基的胞质区域与有丝分裂原活化蛋白激酶（MAPK）的关系，以研究白血病细胞增殖障碍的机制。方法：我们制备了具有互换的细胞质类似物（190 / 130、130 / 190）的嵌合受体，使其在HL-60膜上表达，然后将LIF与野生型受体竞争结合。

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《Journal of Medical Colleges of PLA》 |2001年第3期|p.211-213|共3页
作者
LIU Hou-qi; SHAN Ji-kuan; TANG Shu-ping;
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原文格式 PDF
正文语种 eng
中图分类医药、卫生;
关键词
cytokine; receptor; mitogen-activated protein kinase;

机译：细胞因子受体丝裂原激活的蛋白激酶;

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1. The effect of cytoplasmic regions of LIF receptor on activating MAPK p42/44 in HL-60 cell [J] . LIU Hou-qi, SHAN Ji-kuan, TANG Shu-ping, 中国人民解放军军医大学学报（英文版） . 2001,第003期

机译：LIF受体胞质区对HL-60细胞激活MAPK p42 / 44的影响
2. Involving of the cytoplasmic region of leukemia inhibitory factor receptor alpha subunit, IL-6 related signal transducer-gp130 or fas death domain for MAPK p42/44 activation in HL-60 cell with LIF or anti-Fas IgG. [J] . Liu H, Dan J, Tang S, Molecular and Cellular Biochemistry: An International Journal for Chemical Biology . 2001,第1a2期

机译：白血病抑制因子受体α亚基，IL-6相关信号转导子-gp130或fas死亡结构域的细胞质区域参与LIF或抗Fas IgG激活HL-60细胞中MAPK p42 / 44的作用。
3. Critical role of p42/44(MAPK) activation in anisomycin and hepatocyte growth factor-induced LDL receptor expression: activation of Raf-1/Mek-1/p42/44(MAPK) cascade alone is sufficient to induce LDL receptor expression. [J] . Dhawan P, Bell A, Kumar A, Journal of Lipid Research . 1999,第10期

机译：p42 / 44（MAPK）激活在茴香霉素和肝细胞生长因子诱导的LDL受体表达中的关键作用：单独激活Raf-1 / Mek-1 / p42 / 44（MAPK）级联足以诱导LDL受体表达。
4. Study on Apoptosis of Human promyelocytic leukemia HL-60 Cells Induced by Fucosterol via Death Receptor Pathway [C] . Yu-bin JI, Dong-xue SONG, Chen-feng JI International Conference on Advanced Engineering Materials and Architecture Science . 2013

机译：病毒甾醇通过死亡受体途径诱导的人幼胞细胞白血病HL-60细胞凋亡的研究
5. A systems biology study to delineate the T cell receptor-activated Erk-MAPK signaling pathway. [D] . Zheng, Yanan. 2005

机译：一项系统生物学研究，描述了T细胞受体激活的Erk-MAPK信号通路。
6. Estrogen receptor beta (ERβ) subtype-specific ligands increase transcription p44/p42 mitogen activated protein kinase (MAPK) activation and growth in human non-small cell lung cancer cells [O] . Pamela A. Hershberger, Laura P. Stabile, Beatriz Kanterewicz, -1

机译：雌激素受体β（ERβ）亚型特异性配体增加转录P44 / P42丝裂原活化蛋白激酶（MAPK）活化和人类非小细胞肺癌细胞的生长
7. Genetic deletion of PKR abrogates TNF-induced activation of IκBα kinase, JNK, Akt and cell proliferation but potentiates p44/p42 MAPK and p38 MAPK activation [O] . Y Takada, H Ichikawa, A Pataer, 2006

机译：PKR的遗传缺失废除TNF诱导的IκBα激活激活，JNK，AKT和细胞增殖，但增强剂P44 / P42 MAPK和P38 MAPK激活

The effect of cytoplasmic regions of LIF receptor on activating MAPK p42/44 in HL-60 cell

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