机译:DNA甲基化直接使非CpG岛启动子沉默基因并建立核小体占据的启动子
Department of Pharmacology and Pharmaceutical Sciences, School of Pharmacy,|Department of Urology, Norris Comprehensive Cancer Center, Keck School of Medicine and;
Department of Urology, Norris Comprehensive Cancer Center, Keck School of Medicine and;
Department of Urology, Norris Comprehensive Cancer Center, Keck School of Medicine and;
Department of Pathology, Norris Comprehensive Cancer Center, Keck School of Medicine, University of Southern California, Los Angeles, CA 90033, USA;
Department of Urology, Norris Comprehensive Cancer Center, Keck School of Medicine and;
机译:DNA甲基化可直接使具有非CpG岛启动子的基因沉默,并建立一个核小体占据的启动子。
机译:非CpG岛启动子的DNA甲基化抑制大鼠砷转化的肺上皮细胞中NQO1表达
机译:亨廷顿蛋白调节转录,在体内占据基因启动子,并以多谷氨酰胺依赖性方式直接与DNA结合。
机译:动态核小体定位的功能性转录因子结合位点在诱导型NF-κB靶基因的启动子中
机译:在P19胚胎癌细胞分化过程中,5-羟色胺受体2C基因启动子中CpG岛上DNA甲基化的可能作用。
机译:DNA甲基化直接使非CpG岛启动子沉默基因并建立核小体占据的启动子
机译:DNa甲基化直接用非CpG岛启动子沉默基因并建立核小体占据的启动子