机译:D-柠檬烯通过激活AMPK途径保护PC12细胞免受皮质酮诱导的神经毒性
Huaqiao Univ, Dept Chem & Pharmaceut Engn, Coll Chem Engn, 668 Jimei Ave, Xiamen, Fujian, Peoples R China;
Huaqiao Univ, Dept Chem & Pharmaceut Engn, Coll Chem Engn, 668 Jimei Ave, Xiamen, Fujian, Peoples R China;
Huaqiao Univ, Dept Chem & Pharmaceut Engn, Coll Chem Engn, 668 Jimei Ave, Xiamen, Fujian, Peoples R China;
Huaqiao Univ, Dept Chem & Pharmaceut Engn, Coll Chem Engn, 668 Jimei Ave, Xiamen, Fujian, Peoples R China;
D-Limonene; AMP-activated protein kinase; PC12 cells; Neuroprotection;
机译:D-柠檬烯通过激活AMPK途径来保护PC12细胞免受皮质酮诱导的神经毒性
机译:通过激活AMPK / SIRT1,MAPK / ERK和PI3K / AKT信号通路,通过激活AMPK / SIRT1,MAPK / ERK和信号传导途径来保护PC12细胞免受顺铂诱导的神经毒性的影响
机译:( - ) - EpigallocateChin-3-gallate通过刺猬信号通路保护PC12细胞诱导皮质酮诱导的神经毒性
机译:来自杜仲叶的三种单萜类烯醇免受PC12细胞中的神经毒性
机译:神经元PC12细胞和皮质神经元的凋亡细胞死亡:通过cGMP的融合途径和细胞周期抑制
机译:(-)-Epigallocatechin-3-gallate可通过刺猬信号通路保护PC12细胞免受皮质酮诱导的神经毒性
机译:( - ) - Epigallocatechin-3-gallate通过刺猬信号通路保护PC12细胞免受皮质酮诱导的神经毒性