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机译:NF-κB过度激活会损害胰岛素敏感性,并介导棕榈酸酯诱导的C2C12骨骼肌细胞胰岛素抵抗
Department of Endocrinology, The Second Affiliated Hospital, Sun Yat-Sen University, Guangzhou, 510120, People’s Republic of China;
Department of Endocrinology, Guangdong General Hospital, Guangzhou, 510080, China;
Research Centre, Guangdong General Hospital, Guangzhou, 510080, China;
Department of Endocrinology, The Second Affiliated Hospital, Sun Yat-Sen University, Guangzhou, 510120, People’s Republic of China;
Department of Endocrinology, The Second Affiliated Hospital, Sun Yat-Sen University, Guangzhou, 510120, People’s Republic of China;
Small Interfering RNA; Skeletal muscle; Insulin resistance; Palmitate; NF-kappa B; I-kappa B;
机译:NF-κB过度活化会损害胰岛素敏感性,并介导C2C12骨骼肌细胞中棕榈酸酯诱导的胰岛素抵抗。
机译:使用Platycodon Grandiflorum种子提取物改善C2C12骨骼肌细胞中CALIN诱导的胰岛素抗性
机译:蛋白质酪氨酸磷酸酶-1B(PTP-1B)组合可改善棕榈酸诱导的C2C12骨骼肌细胞中的胰岛素抵抗。
机译:胰岛素快速刺激后L6骨骼肌细胞膜间室向血浆膜的动态变化定量分析
机译:涉及诱导高活性抗逆转录病毒疗法(HAART)的细胞内机制-诱导骨骼肌胰岛素抵抗和提高胰岛素敏感性的潜在治疗方法。
机译:棕榈酸酯诱导的骨骼肌胰岛素抵抗不需要NF-κB激活
机译:棕榈酸酯诱导的骨骼肌胰岛素抵抗不需要NF-B激活。