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MiR-182 affects renal cancer cell proliferation, apoptosis, and invasion by regulating PI3K/AKT/mTOR signaling pathway

机译:MiR-182通过调节PI3K / AKT / MTOR信号传导途径影响肾癌细胞增殖,细胞凋亡和侵袭

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Since this article has been suspected of research misconduct and the corresponding authors did not respond to our request to prove originality of data and figures, “MiR-182 affects renal cancer cell proliferation, apoptosis, and invasion by regulating PI3K/AKT/mTOR signaling pathway, by J.-H. Fu, S. Yang, C.-J. Nan, C.-C. Zhou, D.-Q. Lu, S. Li, H.-Q. Mu, published in Eur Rev Med Pharmacol Sci 2018; 22 (2): 351-357–DOI: 10.26355/eurrev_201801_14179–PMID: 29424922” has been withdrawn. The Publisher apologizes for any inconvenience this may cause. MiR-182 affects renal cancer cell proliferation, apoptosis, and invasion by regulating PI3K/AKT/mTOR signaling pathway.
机译:由于本文被疑似研究不当行为,并且相应的作者没有回应我们证明数据和数字的原创性的要求,“MiR-182通过调节PI3K / AKT / MTOR信号传导途径来影响肾癌细胞增殖,细胞凋亡和侵袭,由J-H.傅,S.杨,C.-J。 Nan,C.-c。周,D.-q.鲁,李,李,H.-q.穆,发表于欧元兑欧元医药SCI 2018; 22(2):351-357-DOI:10.26355 / EURREV_201801_14179-PMID:29424922“已被撤回。出版商对由此带来的任何不便表示歉意。 MiR-182通过调节PI3K / AKT / MTOR信号传导途径来影响肾癌细胞增殖,细胞凋亡和侵袭。

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