Involvement of Oct4 in the pathogenesis of thoracic aortic dissection via inducing the dedifferentiated phenotype of human aortic smooth muscle cells by directly upregulating KLF5

Yan Yan; Meng-Wei Tan; Xiang Xue; Xue-Yan Ding; Guo-Kun Wang; Zhi-Yun Xu

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Involvement of Oct4 in the pathogenesis of thoracic aortic dissection via inducing the dedifferentiated phenotype of human aortic smooth muscle cells by directly upregulating KLF5

机译：通过直接上调KLF5诱导人主动脉平滑肌细胞的去分化表型，Oct4参与胸主动脉夹层的发病机制

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《The journal of thoracic and cardiovascular surgery》 |2016年第3期|共10页
作者
Yan Yan; Meng-Wei Tan; Xiang Xue; Xue-Yan Ding; Guo-Kun Wang; Zhi-Yun Xu;
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1. Involvement of Oct4 in the pathogenesis of thoracic aortic dissection via inducing the dedifferentiated phenotype of human aortic smooth muscle cells by directly upregulating KLF5 [J] . Yan Yan, Tan Meng-Wei, Xue Xiang, The Journal of Thoracic and Cardiovascular Surgery . 2016,第3期

机译：通过直接上调KLF5诱导人主动脉平滑肌细胞的去分化表型，Oct4参与胸主动脉夹层的发病机制
2. Association of smooth muscle cell phenotypes with extracellular matrix disorders in thoracic aortic dissection [J] . WangL., ZhangJ., FuW., Journal of vascular surgery . 2012,第6期

机译：胸主动脉夹层中平滑肌细胞表型与细胞外基质疾病的关系
3. TGFBR2 mutations alter smooth muscle cell phenotype and predispose to thoracic aortic aneurysms and dissections. [J] . Inamoto S, Kwartler CS, Lafont AL, Cardiovascular Research . 2010,第3期

机译：TGFBR2突变会改变平滑肌细胞表型，并易患胸主动脉瘤和解剖。
4. Aortic smooth muscle cells respond to soluble proteins released by aortic endothelial cells after fluid flow exposure [C] . McCann, J.A., Webster, . 2002

机译：暴露于流体流后，主动脉平滑肌细胞对主动脉内皮细胞释放的可溶性蛋白质有反应
5. Vascular smooth muscle Sirtuin-1 protects against aortic dissection during angiotensin II-induced hypertension. [D] . Akiki, Rachid. 2016

机译：血管平滑肌Sirtuin-1可防止血管紧张素II诱发的高血压时的主动脉夹层。
6. TGFBR2 mutations alter smooth muscle cell phenotype and predispose to thoracic aortic aneurysms and dissections [O] . Sakiko Inamoto, Callie S. Kwartler, Andrea L. Lafont, -1

机译：TGFBR2突变改变平滑肌细胞表型并易患胸主动脉瘤和解剖
7. Involvement of Oct4 in the pathogenesis of thoracic aortic dissection via inducing the dedifferentiated phenotype of human aortic smooth muscle cells by directly upregulating KLF5 [O] . Yan Yan, Meng-Wei Tan, Xiang Xue, 2016

机译：10.直接上调KLF5，OCT4通过诱导人主动脉平滑肌细胞的消除表型胸腔主动脉夹层的发病机制

Involvement of Oct4 in the pathogenesis of thoracic aortic dissection via inducing the dedifferentiated phenotype of human aortic smooth muscle cells by directly upregulating KLF5

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