机译:KAI1 / CD82介导的癌症侵袭和转移抑制需要跨膜相互作用。
From the Vascular Biology Center, Center for Cancer Research, and Departments of Medicine and Molecular Science,University of Tennessee Health Science Center, Memphis, Tennessee;
From the Vascular Biology Center, Center for Cancer Research, and Departments of Medicine and Molecular Science,University of Tennessee Health Science Center, Memphis, Tennessee;
From the Vascular Biology Center, Center for Cancer Research, and Departments of Medicine and Molecular Science,University of Tennessee Health Science Center, Memphis, Tennessee;
the Department of Structural Biology,St. Jude Children’s Research Hospital, Memphis, Tennessee;
and the Department of Biological Sciences,University of Iowa, Iowa City, Iowa;
the Department of Structural Biology,St. Jude Children’s Research Hospital, Memphis, Tennessee;
From the Vascular Biology Center, Center for Cancer Research, and Departments of Medicine and Molecular Science,University of Tennessee Health Science Center, Memphis, Tennessee;
机译:跨膜相互作用对于KAI1 / CD82介导的癌症侵袭和转移抑制是必需的。
机译:肿瘤细胞上的KAI1与血管内皮上的DARC的相互作用导致转移抑制。
机译:Kai1基因通过人前列腺癌的ATF3-NFκB络合物从事NDRG1基因介导的转移抑制
机译:3D玻璃纳米流体由飞秒激光加工制造,用于研究癌细胞转移和侵袭
机译:KAI1,人类乳腺癌的转移抑制基因。
机译:KAI1 / CD82介导的癌症侵袭和转移抑制需要跨膜相互作用。
机译:KAI1 / CD82介导的癌症侵袭和转移抑制需要跨膜相互作用。
机译:纤溶酶 - 细胞相互作用在乳腺癌侵袭和转移中的作用