Early neutrophil infiltration is critical for inflammation-sensitized hypoxic-ischemic brain injury in newborns

机译：早期中性粒细胞浸润对于新生儿炎症敏感的缺氧缺血性脑损伤至关重要

代理获取

本网站仅为用户提供外文OA文献查询和代理获取服务，本网站没有原文。下单后我们将采用程序或人工为您竭诚获取高质量的原文，但由于OA文献来源多样且变更频繁，仍可能出现获取不到、文献不完整或与标题不符等情况，如果获取不到我们将提供退款服务。请知悉。

页面导航

著录项
引文网络
相似文献
相关主题

著录项

期刊名称 Journal of Cerebral Blood Flow Metabolism
作者
Hui-Wen Yao; Chia-Yi Kuan;
展开▼
作者单位

展开▼
年(卷),期 2019(40),11
年度 2019
页码 2188–2200
总页数 13
原文格式 PDF
正文语种
中图分类神经科学;血液学检验;
关键词

机译：缺氧缺血性脑病;中性粒细胞细胞外陷阱;脂多糖;基质金属蛋白酶;抗LY6G;

相似文献

外文文献
中文文献
专利

1. Blocking Lymphocyte Trafficking with FTY720 Prevents Inflammation-Sensitized Hypoxic-Ischemic Brain Injury in Newborns [J] . Yang Dianer, Sun Yu-Yo, Bhaumik Siddhartha Kumar, The Journal of Neuroscience: The Official Journal of the Society for Neuroscience . 2014,第49期

机译：用FTY720阻止淋巴细胞贩运可预防炎症敏感的新生儿缺氧缺血性脑损伤
2. Inter-Alpha Inhibitor Proteins (IAIPs) Reduce Neutrophilic Infiltration into Brain and Relative Increases in Systemic Neutrophils in Neonatal Rats after Hypoxic-Ischemic (HI) Brain Injury [J] . Barrios-Anderson A., Chen X., Nakada S., The Journal of head trauma rehabilitation . 2018,第3期

机译：α间抑制剂蛋白（IAIPS）将中性粒乳浸润降至脑缺氧（HI）脑损伤后新生大鼠的全身中性粒细胞相对增加
3. Inter-Alpha Inhibitor Proteins (IAIPs) Reduce Neutrophilic Infiltration into Brain and Relative Increases in Systemic Neutrophils in Neonatal Rats after Hypoxic-Ischemic (HI) Brain Injury [J] . Barrios-Anderson A., Chen X., Nakada S., The Journal of head trauma rehabilitation . 2018,第3期

机译：α间抑制剂蛋白（IAIPS）将中性粒乳浸润降至脑缺氧（HI）脑损伤后新生大鼠的全身中性粒细胞相对增加
4. The Relationship between the Newborn Rats' Hypoxic-Ischemic Brain Damage and Heart Beat Interval Information [C] . Xiaomin Jiang, Hiroki Tamura, Koichi Tanno, International conference on neural information processing;ICONIP 2011 . 2011

机译：新生大鼠缺氧缺血性脑损伤与心跳间隔信息的关系
5. Hyperhomocysteinemia Accelerates Stroke-Induced Brain Injury Via Promoting Endothelial Activation and Inflammatory Cell Infiltration: The Role of ICAM1-Mediated Neutrophil and Monocyte Infiltration [D] . Zhang, Lixiao. 2017

机译：高同型半胱氨酸血症通过促进内皮细胞活化和炎性细胞浸润加速中风诱发的脑损伤：ICAM1介导的中性粒细胞和单核细胞浸润的作用
6. Involvement of CXCL1/CXCR2 During Microglia Activation Following Inflammation-Sensitized Hypoxic-Ischemic Brain Injury in Neonatal Rats [O] . Meray Serdar, Karina Kempe, Ralf Herrmann, 2020

机译：在新生大鼠炎症敏化症状缺氧缺血性脑损伤后CXCL1 / CXCR2在新生大鼠缺氧缺血性脑损伤期间参与
7. Blocking lymphocyte trafficking with FTY720 prevents inflammation-sensitized hypoxic-ischemic brain injury in newborns [O] . Yang, D 2015

机译：用FTY720阻止淋巴细胞运输可防止炎症敏感的新生儿缺氧缺血性脑损伤
8. Increased Expression of the Chemokines CXCL1 and MIP-1a by Resident Brain Cells Precedes Neutrophil Infiltration in the Brain Following Prolonged Soman-Induced Status Epilepticus in Rats [R] . Johnson, E. A., Dao, T. L., Guignet, M. A., 2011

机译：驻留脑细胞增加趋化因子CXCL1和mIp-1a的表达在大鼠中长期梭曼诱导的癫痫持续状态后中性粒细胞浸润

Early neutrophil infiltration is critical for inflammation-sensitized hypoxic-ischemic brain injury in newborns

著录项

引文网络

相似文献

相关主题

期刊订阅