A method for the production of hollow bodies made of plastic press masses for the apparatus

• 1. 由热塑性塑料制造空心体的方法和由热塑性塑料制造空心体的设备 [P] . 中国专利： CN102056726B . 2016.03.16
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  机译： 用于生产由纤维素块和类似塑料块制成的空心体的设备

• 4. Method and apparatus for the production of hollow bodies made of plastic masses [P] . 德国专利： DE695454C . 1940-08-26

  机译： 用于制造由塑料块制成的空心体的方法和设备

• 5. Centrifugal method and apparatus for the production of hollow bodies of concrete or similar plastic masses [P] . 德国专利： DE531940C . 1931-08-20

  机译： 用于生产混凝土或类似塑料块的空心体的离心方法和设备

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• 1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 2. Compressive and Fracture Properties of Syntactic Foam Filled with Hollow Plastic Bead(HPC) [J] . 谢文锋 . 武汉理工大学学报：材料科学英文版 . 2007,第3期
• 3. 我国真空设备行业制造的主要真空获得设备概况与思考 [C] . 于溥 . 2005’全国真空冶金与表面工程学术会议 . 2005
• 4. 一种基于实心体素化的实时渲染方法研究 [A] . 杨柳青 . 2014