Method and apparatus for minimizing cavity effects in acoustic logging

• 1. 用于在真空腔室中成像的设备、用于真空处理基板的系统、和用于使真空腔室中的至少一个对象成像的方法 [P] . 中国专利： CN109891618B . 2021.02.26
• 2. 用于在真空腔室中成像的设备、用于真空处理基板的系统、和用于使真空腔室中的至少一个对象成像的方法 [P] . 中国专利： CN109891618A . 2019-06-14
• 3. METHOD AND APPARATUS FOR MINIMIZING CAVITY EFFECTS IN ACOUSTIC LOGGING [P] . CA717202A . 1965-08-31

  机译： 最小化声波测井中空洞效应的方法和装置

• 4. METHOD AND APPARATUS FOR MINIMIZING FAR-END SPEECH EFFECTS IN HANDS-FREE TELEPHONY SYSTEMS USING ACOUSTIC BEAMFORMING [P] . CA2445180C . 2007-02-06

  机译： 利用声学波束成形来最小化无手电话系统中的远端语音效果的方法和装置

• 5. Method and apparatus for minimizing far-end speech effects in hands-free telephony systems using acoustic beamforming [P] . 英国专利： GB2367730B . 2005-04-27

  机译： 使用声波束成形在免提电话系统中最小化远端语音效果的方法和装置

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• 1. Analysis on Physical Mechanism of Sound Generation inside Cavities Based on Acoustic Analogy Method [J] . Dangguo Yang ,Jianqiang Li ,Jun Liu . 流体动力学（英文） . 2013,第1期
• 2. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 3. 飞机设备舱门开启空腔流动效应下舵面振动响应分析方法研究 [C] . HUANG Hu ,黄虎 ,JIN Wei . 四川省力学学会2014年学术交流年会 . 2014
• 4. 空腔和堆积型多孔介质中瑞利--贝纳德对流的热脉动效应研究 [A] . 赵俊龙 . 2019