Method and apparatus for separating sheet packets containing defects or marked by adhesive sheets of paper or the like.

• 1. 用于自动识别产品的产品缺陷和/或用于自动识别产品缺陷的产品缺陷原因的方法和设备 [P] . 中国专利： CN113396444A . 2021-09-14
• 2. 用于在制备分离器活性层之前定量构成分离器活性层的胺化合物的方法、用于定量分离器活性层中的聚酰胺或未反应的胺化合物的方法、以及用于设定分离器活性层的制备条件或用于分离器活性层的制备条件的确立标准的方法 [P] . 中国专利： CN111566473A . 2020-08-21
• 3. Plastic cover for binding of a packet of paper sheets by means of thermosetting adhesive and method for manufacturing of a plastic cover for binding of a packet of paper sheets by means of thermosetting adhesive [P] . PL420307A1 . 2018-07-30

  机译： 通过热固性粘合剂粘合纸包的塑料盖和制造通过热固性粘合剂粘合纸包的塑料盖的方法

• 4. PAPER SHEET SEPARATING DEVICE, PAPER SHEET SEPARATING TAKEOUT DEVICE, PAPER SHEET TREATMENT DEVICE, PAPER SHEET SEPARATING METHOD AND PAPER SHEET SEPARATING TAKEOUT METHOD [P] . 日本专利： JP2008222367A . 2008-09-25

  机译： 纸片分离装置，纸片分离取出装置，纸片处理装置，纸片分离方法和纸片分离取出方法

• 5. SEPARABLE SHEET SEPARATING METHOD AND DEVICE FOR PRESSURE-SENSITIVE ADHESIVE PAPER SHEET FITTED WITH SEPARABLE SHEET [P] . 日本专利： JPH08175744A . 1996-07-09

  机译： 装有可分离片材的压敏胶纸的可分离片材分离方法和装置

• 1. Experimental research on refractory performance of reinforced concrete beams strengthened with CFRP sheets bonded with an inorganic adhesive [J] . 郑文忠, 万夫雄, 李时光 哈尔滨工业大学学报：英文版 . 2010,第004期
• 2. Experimental research on refractory performance of reinforced concrete beams strengthened with CFRP sheets bonded with an inorganic adhesive [J] . ZHENG Wen-zhong, WAN Fu-xiong, LI Shi-guang 哈尔滨工业大学学报（英文版） . 2010,第004期
• 3. Influence of pre-generated infinite adhesive defects on the forming behaviour of adhesive bonded steel sheets [C] . V. Satheeshkumar, R. Ganesh Narayanan International Conference on Advanced Materials and Processing Technologies . 2014
• 4. Characterization of Galvanized/Galvannealed Sheet Steel Defects Towards Enabling Defect Free Zinc Coatings [D] . Plessinger, Ryan. 2019
• 5. Effect of the Application of Different Surface Treatment Methods on the Strength of Titanium Alloy Sheet Adhesive Lap Joints [O] . Anna Rudawska, Kazimierz Zaleski, Izabela Miturska, 2019
• 6. Quantitative Estimation of Sheet Formation of Insulating Paper and Effect of Sheet Formation on the Dielectric Breakdown of the Oil-Impregnated Paper [O] . M. MURATA, F. NAKATANI, K. NAKADA 1972
• 7. Nonlinear Adhesive Behavior Effects in a Cracked Orthotropic Sheet Stiffened by a Semi-Infinite Orthotropic Sheet [R] . Bigelow, C. A. 1985

• 1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 2. Springback of Aircraft Sheet Metal Hydro-Pressure and Stretch Forming Methods [J] . Akira Kono ,Sousuke Sasaki ,Susumu Takahashi . 材料科学与工程：中英文A版 . 2014,第009期
• 3. New Methods Testing of Adhesion of the Coating to Sheet Metal by Bending [J] . Milan Dvorak ,Emil Schwarzer . 表面工程材料与先进技术期刊（英文） . 2012,第2期
• 4. YB55小盒透明纸包装机烟库料位检测缺陷分析及改进方法 [C] . 祝荣壮 . 广西烟草学会2007年度学术年会 . 2008
• 5. 天然生物源何帕烷三萜类化合物及其同位素标记类似物的合成方法研究 [A] . 潘卫东 . 2007