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Extensor deficiency: first cause of childhood flexible flat foot.

机译:伸肌缺乏症:儿童期导致足底柔韧的首要原因。

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摘要

Childhood flexible flat foot is the most common paramorphism of the lower limb. The cause is not a bony malformation of the foot but a functional deficiency of the anatomic structures supporting the plantar arch. These structures, working as active tie rods (the tibialis anterior and posterior muscles) or passive factors of support (flexor hallucis longus and flexor digitorum longus muscles) act together to maintain the plantar arch. Their deficiency is responsible for childhood flexible flat foot, characterized by a flattening of the plantar arch and calcaneus pronation (heel valgus) and manifested in the characteristic "duck walking" in children. Hypothesizing poor extensor activity of the tibialis anterior, extensor digitorum longus, and extensor hallucis longus muscles during the heel contact phase of the gait cycle, we began a preliminary study to evaluate, through superficial electromyography (sEMG), the activation of muscle groups involved in the pathogenesis of childhood flexible flat foot, in particular the tibialis anterior and extensor hallucis longus muscles, to plan a rehabilitative program addressing the strengthening of insufficient muscles. The therapeutic program should also include the use of a medial elastic push orthosis. Data obtained by sEMG highlight a reduced activation of muscles related to the grade of flat foot, emphasizing the concept that a reduced activation of extensor muscles may be involved in determining flexible flat foot.
机译:儿童期灵活的扁平足是下肢最常见的变形。原因不是脚的骨畸形,而是支撑足弓的解剖结构的功能缺陷。这些结构充当主动横拉杆(胫骨前肌和后肌)或被动支撑因子(拇长屈肌和趾长屈肌)共同作用以维持maintain弓。他们的缺陷是造成儿童期柔软的扁平足的原因,其特征是足弓和跟骨前旋(足跟外翻)变平,并表现为儿童特征性的“鸭子行走”。假设步态周期的足跟接触阶段胫骨前,伸指长肌和拇长伸肌的伸肌活动不良,我们开始了一项初步研究,以通过浅表肌电图(sEMG)评估参与该过程的肌肉群的激活儿童软足扁平足的发病机理,尤其是胫前肌和拇长伸肌,以制定康复计划,以解决肌肉不足的问题。治疗程序还应包括使用内侧弹性推入矫形器。 sEMG获得的数据突出显示了与扁平足等级有关的肌肉激活减少,强调了在确定柔性扁平足中可能涉及到伸肌激活减少的概念。

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