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首页> 外文期刊>Catheterization and cardiovascular interventions: Official journal of the Society for Cardiac Angiography & Interventions >Abnormal distortion of aortic corevalve bioprosthesis with suicide left ventricle, aortic insufficiency, and severe mitral regurgitation during transcatheter aortic valve replacement
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Abnormal distortion of aortic corevalve bioprosthesis with suicide left ventricle, aortic insufficiency, and severe mitral regurgitation during transcatheter aortic valve replacement

机译:经导管主动脉瓣置换术中自杀性左心室主动脉瓣生物瓣膜异常变形,主动脉瓣关闭不全和严重二尖瓣反流

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We present a patient with critical degenerative aortic stenosis, mitral annular and aortomitral continuity calcification, and senile sigmoid septal hypertrophy who underwent transcatheter aortic valve replacement using the CoreValve bioprosthesis. Immediately after predilation of the aortic valve (18-mm balloon), the patient developed severe hypotension and dynamic left ventricular outflow tract (LVOT) obstruction with systolic anterior motion of the anterior mitral leaflet, causing severe mitral regurgitation. After deployment of a 26-mm bioprosthesis, a transesophageal echocardiogram and left ventriculogram showed that the frame of the bioprosthesis appeared distorted and underexpanded. On the mitral side of the aorta (side of the aortomitral curtain between 12:00 and 3:00, echo short axis view), we found moderate periprosthetic aortic insufficiency with worse mitral regurgitation. The left ventricle was small and hyperdynamic (ejection fraction >85%). The patient soon developed complete heart block, atrial fibrillation, and ventricular tachycardia. She was resuscitated with aggressive intravenous fluids, vasopressors, and an emergently placed atrioventricular sequential pacemaker. We postdilated the 26-mm bioprosthesis with a 22-mm Z-Med balloon and subsequently with a 25-mm balloon. Each balloon was inflated to its nominal volume and pressure and conformed the nitinol frame of the valve to the net circular shape and expected diameter. However, as soon as each balloon was deflated, the surrounding aortic root anatomy visibly recoiled and the frame returned to its smaller diameter with a distorted shape. A second 26-mm CoreValve bioprosthesis was then deployed in a valve-in-valve configuration. Soon after, the patient's hemodynamics improved, her clinical condition stabilized, and she completely recovered. (c) 2016 Wiley Periodicals, Inc.
机译:我们介绍了患有严重的退化性主动脉瓣狭窄,二尖瓣环和主动脉连续性钙化以及老年乙状结肠间隔肥大的患者,他们使用CoreValve生物瓣膜进行了经导管主动脉瓣置换术。主动脉瓣扩张(18毫米球囊)扩张后,患者立即出现严重的低血压和动态左心室流出道(LVOT)阻塞,并伴有二尖瓣前叶的收缩前运动,从而导致严重的二尖瓣反流。部署26 mm的生物假体后,经食道超声心动图和左心室图显示该生物假体的框架出现扭曲和扩张不足。在主动脉的二尖瓣一侧(在12:00和3:00之间的主动脉瓣幕的一侧,回声短轴视图),我们发现中度假体周围主动脉瓣关闭不全,二尖瓣反流不良。左心室小且运动亢进(射血分数> 85%)。患者很快出现了完全性的心脏传导阻滞,房颤和室性心动过速。用积极的静脉输液,血管加压药和急需放置的房室顺序起搏器使她复苏。我们将22毫米Z型Med球囊扩张到26毫米生物假体中,然后再使用25毫米球囊扩张。将每个球囊充气至其标称体积和压力,并使瓣膜的镍钛合金框架符合净圆形形状和预期直径。但是,每个气囊放气后,周围的主动脉根部解剖结构都会明显缩回,并且镜架恢复其较小的直径,并且形状变形。然后将第二个26毫米CoreValve生物假体部署为瓣膜阀配置。此后不久,患者的血流动力学改善,临床状况稳定,完全康复。 (c)2016年威利期刊有限公司

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