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首页> 外文期刊>British Journal of Radiology >Non-tumorous enhancement caused by cholecystic venous inflow shown on biphasic CT hepatic arteriography: comparison with hepatocellular carcinoma.
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Non-tumorous enhancement caused by cholecystic venous inflow shown on biphasic CT hepatic arteriography: comparison with hepatocellular carcinoma.

机译:双相CT肝动脉造影显示胆囊静脉流入引起的非肿瘤性增强:与肝细胞癌的比较。

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The haemodynamics in non-tumorous abnormalities on CT arterial portography (CTAP) owing to cholecystic venous direct inflow to the liver were compared with the haemodynamics in hepatocellular carcinoma. 53 patients who simultaneously underwent CTAP and CT during hepatic arteriography (CTHA) to detect hepatocellular carcinoma had the late phase added to CTHA. Changes in size, shape and pattern of 47 non-tumorous enhancement abnormalities on the liver around the gall bladder or in the dorsum of segment IV between the early and late phases on biphasic CTHA as well as of 60 tumorous lesions were determined. Enhancement on biphasic CTHA was seen in all 47 lesions with a non-tumorous portal defect (early phase alone, n=8; late phase alone, n = 3; both, n = 36). In these 47 lesions, the size and the shape of enhancement changed in 63.8% and 51.1%, respectively, between the early and late phases on CTHA; the pattern of enhancement did not change in 72.3%. On the other hand, the size of enhancement on biphasic CTHA changed in only 16.7% of 60 tumours, and the shape in only 5%, although the enhancement pattern changed in a large proportion (80%). In conclusion, owing to the difference in haemodynamics, non-tumorous abnormalities caused by cholecystic venous inflow and tumours are clearly delineated on biphasic CTHA. Thus, adding the late phase to previous single phase CTHA (i.e. performing biphasic CTHA) is useful in differentiating the two entities.
机译:将由于胆囊静脉直接流入肝脏而导致的CT动脉门静脉造影(CTAP)非肿瘤异常的血液动力学与肝细胞癌的血液动力学进行了比较。 53例同时在肝动脉造影(CTHA)期间接受CTAP和CT检查以检测肝细胞癌的患者在CTHA中增加了晚期。确定了双相CTHA早期和晚期之间胆囊周围肝脏或IV段背侧47种非肿瘤性增强异常的大小,形状和模式的变化,以及60种肿瘤性病变的变化。在所有47例具有非肿瘤性门静脉缺损的病变中均观察到双相CTHA增强(单独早期,n = 8;单独晚期,n = 3;两者,n = 36)。在这47个病变中,CTHA的早期和晚期之间,增强的大小和形状分别改变了63.8%和51.1%。增强模式没有变化,为72.3%。另一方面,尽管增强模式变化很大(80%),但双相CTHA增强的大小在60个肿瘤中仅改变了16.7%,形状仅改变了5%。总之,由于血液动力学的差异,在双相CTHA上清楚地描述了由胆囊静脉流入和肿瘤引起的非肿瘤异常。因此,将后期添加到先前的单相CTHA(即执行双相CTHA)可用于区分两个实体。

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