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机译:TCR诱导的PKC-θ介导的NF-κB活化由Caspase-8-胱天蛋白酶-9-胱天蛋白-3级联调节
Department of Cardiology Xiangya Hospital Central South University Hunan 41000 China;
Section of Nephrology Department of Medicine University of Chicago IL 60637 United States;
Section of Nephrology Department of Medicine University of Chicago IL 60637 United States;
Section of Nephrology Department of Medicine University of Chicago IL 60637 United States;
Department of Cardiology Xiangya Hospital Central South University Hunan 41000 China;
Section of Nephrology Department of Medicine University of Chicago IL 60637 United States;
Caspase-9; NF-κB; T cell activation;
机译:TCR诱导的PKC-θ介导的NF-κB活化由Caspase-8-胱天蛋白酶-9-胱天蛋白-3级联调节
机译:NEMO差异性调节TCR和TNF-α诱导的NF-κB通路,并在TCR诱导的NF-κB激活中具有抑制作用
机译:USP2a通过桥接MALT1-TRAF6积极调节TCR诱导的NF-κB活化
机译:Toll样受体2介导的生物材料表面的损伤相关分子模式激活NF-κB
机译:布鲁顿酪氨酸激酶通过B细胞受体和BAFF-R调节NF-κB活化和B细胞存活
机译:TCR诱导PKC-θ介导的NF-κB活化受Caspase-8-Caspase-9-Caspase-3级联调控。
机译:TCR诱导的PKC-θ介导的NF-κB活化由Caspase-8-胱天蛋白-9-胱天蛋白-3级联调节