机译:姜黄素通过抑制IGF2和IGF2介导的PI3K / AKT / MTOR信号传导途径来抑制尿栓肿瘤发育
Shanghai Jiao Tong Univ Affiliated Peoples Hosp 6 Dept Urol 600 Yishan Rd Shanghai 200233;
Fudan Univ Pudong Med Ctr Shanghai Pudong Hosp Dept Gynaecol &
Obstet Shanghai Peoples R China;
Shanghai Jiao Tong Univ Affiliated Peoples Hosp 6 Dept Urol 600 Yishan Rd Shanghai 200233;
Shanghai Jiao Tong Univ Affiliated Peoples Hosp 6 Dept Urol 600 Yishan Rd Shanghai 200233;
Shanghai Jiao Tong Univ Affiliated Peoples Hosp 6 Dept Urol 600 Yishan Rd Shanghai 200233;
Shanghai Jiao Tong Univ Affiliated Peoples Hosp 6 Dept Urol 600 Yishan Rd Shanghai 200233;
Shanghai Jiao Tong Univ Affiliated Peoples Hosp 6 Dept Urol 600 Yishan Rd Shanghai 200233;
Shanghai Jiao Tong Univ Affiliated Peoples Hosp 6 Dept Urol 600 Yishan Rd Shanghai 200233;
Curcumin; urothelial tumor; IGF2; PI3K/AKT/mTOR;
机译:姜黄素通过抑制IGF2和IGF2介导的PI3K / AKT / MTOR信号传导途径来抑制尿栓肿瘤发育
机译:来自Garcinia Nujiangensis的抗肿瘤X吨数抑制了人类卵巢癌细胞中PARP,PI3K / AKT / MTOR和MAPK / ERK信号通路的诱导细胞凋亡
机译:通过抑制SKOV-3细胞中的EGFR介导的P38 / MAPK和PI3K / AKT / MTOR信号传导途径抑制血管蛋白的硫酸化合物墨水墨水剂抑制迁移,侵袭和基质金属蛋白酶-2表达
机译:内皮细胞和肿瘤细胞中PI3K / Akt / mTOR抑制剂和PDT的组合。
机译:PI3k / AKT / mTOR信号通路的遗传分析。
机译:通过抑制PI3K / Akt / mTOR信号通路MDA19在人骨肉瘤中的抗肿瘤潜力
机译:姜黄素通过MIR-206的上调抑制非小细胞肺癌细胞的迁移和侵袭,抑制PI3K / AKT / MTOR信号通路的抑制