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机译:新型HSP90抑制剂蛋白酶粘接蛋白D破坏HSP90 / CDC37复合物,增强了MTOR抑制剂的抗癌作用
State Key Laboratory of Quality Research in Chinese Medicine Institute of Chinese Medical Sciences;
State Key Laboratory of Quality Research in Chinese Medicine Institute of Chinese Medical Sciences;
College of Pharmaceutical Sciences Zhejiang University;
State Key Laboratory of Quality Research in Chinese Medicine Institute of Chinese Medical Sciences;
College of Pharmaceutical Sciences Zhejiang University;
State Key Laboratory of Quality Research in Chinese Medicine Institute of Chinese Medical Sciences;
State Key Laboratory of Quality Research in Chinese Medicine Institute of Chinese Medical Sciences;
State Key Laboratory of Quality Research in Chinese Medicine Institute of Chinese Medical Sciences;
State Key Laboratory of Natural Medicines China Pharmaceutical University;
State Key Laboratory of Natural Medicines China Pharmaceutical University;
College of Pharmaceutical Sciences Zhejiang University;
State Key Laboratory of Quality Research in Chinese Medicine Institute of Chinese Medical Sciences;
Everolimus; Platycodin D; Hsp90; Cdc37; EGFR; IGF1R;
机译:新型HSP90抑制剂蛋白酶粘接蛋白D破坏HSP90 / CDC37复合物,增强了MTOR抑制剂的抗癌作用
机译:人类Hsp90-p50(Cdc37)伴侣蛋白对核苷酸和Hsp90抑制剂的稳定性,以及酪蛋白激酶2磷酸化的影响。
机译:天然植物类黄酮芹菜素直接破坏Hsp90 / Cdc37复合物并抑制胰腺癌细胞的生长和迁移
机译:HSP90 / CDC37伴侣/共伴侣复合物,采用草药药物的作用方式阐明了一种新的结抗癌目标A.
机译:NQO1在苯醌安沙霉素Hsp90抑制剂的代谢中的作用以及新型Hsp90抑制剂作为抗癌剂的开发。
机译:HSP90抑制剂AUY922通过破坏白血病细胞中的Bcr-AblJak2和HSP90信号网络复合物诱导细胞死亡
机译:AKT与热休克蛋白90(HSP90)和CDC37形成细胞内复合物,并通过HSP90功能的抑制剂稳定