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Metabolic alkalosis due to short bowel syndrome in a hemodialysis patient

机译:由于血液透析患者肠道综合征短肠综合征引起的代谢碱化

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A 53-year-old man on hemodialysis suffered from short bowel syndrome after subtotal colectomy and partial resection of the small intestine. Laboratory tests showed multiple electrolyte disorders and enlarged sodium and chloride ion (Cl-) gaps despite treatment with large volume of sodium chloride replacement via central venous infusion. Blood gas analysis showed slightly high bicarbonate ion levels and metabolic alkalosis was suspected, which is uncommon in end stage kidney disease. The measurement of electrolytes in feces from ileostomy showed relatively high Cl- excretion. The loss of Cl- to feces may have caused the metabolic alkalosis; thus, additional Cl- replacement may have been necessary.
机译:一名53岁的血液透析血液透析后患者患有短肠综合征后畸形综合症和小肠部分切除。 尽管通过中央静脉输注用大量的氯化钠置换治疗,但实验室测试表现出多种电解质障碍和扩大的钠和氯离子(CL-)间隙。 血气分析显示出略微高碳酸氢盐离子水平,怀疑代谢碱中毒,其在最终阶段的肾脏疾病中是罕见的。 来自对鼠瘘的粪便中的电解质的测量显示出相对高的CL-排泄。 Cl-to Feeces的丧失可能导致代谢碱中毒; 因此,可能需要额外的CL-替换。

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