机译:通过激活NGF信号通路,通过激活NGF信号通路来保护PC12细胞免受MPP +毒性保护PC12细胞的保护
Univ Sao Paulo FCFRP Dept Anal Clin Toxicol &
Bromatol Av Cafe S-N BR-14040903 Ribeirao Preto;
Univ Sao Paulo FCFRP Dept Anal Clin Toxicol &
Bromatol Av Cafe S-N BR-14040903 Ribeirao Preto;
Univ Sao Paulo FCFRP Dept Anal Clin Toxicol &
Bromatol Av Cafe S-N BR-14040903 Ribeirao Preto;
Univ Sao Paulo FCFRP Dept Anal Clin Toxicol &
Bromatol Av Cafe S-N BR-14040903 Ribeirao Preto;
Univ Estadual Paulista UNESP Inst Quim Araraquara Araraquara SP Brazil;
Univ Estadual Paulista UNESP Inst Quim Araraquara Araraquara SP Brazil;
Univ Estadual Paulista UNESP Inst Quim Araraquara Araraquara SP Brazil;
Univ Sao Paulo FCFRP Dept Anal Clin Toxicol &
Bromatol Av Cafe S-N BR-14040903 Ribeirao Preto;
Univ Sao Paulo FCFRP Dept Anal Clin Toxicol &
Bromatol Av Cafe S-N BR-14040903 Ribeirao Preto;
Snake-venom peptides; Neuroprotection; Neurotrophin receptors trkA (tropomyosin receptor kinase A); MPP+ (1-methyl-4-phenylpyridinium); PC12 cells;
机译:通过激活NGF信号通路,通过激活NGF信号通路来保护PC12细胞免受MPP +毒性保护PC12细胞的保护
机译:通过激活NGF信号通路,咖啡酸苯乙酯(己酯)通过激活NGF信号传导途径保护来自顺铂诱导的神经毒性的PC12细胞
机译:D-柠檬烯通过激活AMPK途径保护PC12细胞免受皮质酮诱导的神经毒性
机译:来自杜仲叶的三种单萜类烯醇免受PC12细胞中的神经毒性
机译:神经元PC12细胞和皮质神经元的凋亡细胞死亡:通过cGMP的融合途径和细胞周期抑制
机译:利福平通过PERK-eIF2α-ATF4途径激活GRP78保护PC12细胞免受鱼藤酮诱导的细胞毒性
机译:基于蛇毒液的三肽(Glu-Val-TRP)通过激活NGF信号通路来保护PC12细胞免受MPP +毒性的影响