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机译:姜黄素衰减由肿瘤坏死因子-α或异丙肾上腺素刺激的脂肪分解在3T3-L1 adipocytes中
Division of Geriatric Medicine Tongji Hospital Tongji University Shanghai 200065 China;
Division of Endocrinology and Metabolism Shanghai Ninth People's Hospital Shanghai JiaoTong;
Institute of Integrative Medicine Huashan Hospital Fudan University Shanghai 200040 China;
Division of Endocrinology and Metabolism Shanghai Ninth People's Hospital Shanghai JiaoTong;
Division of Endocrinology and Metabolism Shanghai Ninth People's Hospital Shanghai JiaoTong;
Adipocyte; Curcumin; Isoproterenol; Lipolysis; TNFα;
机译:姜黄素衰减由肿瘤坏死因子-α或异丙肾上腺素刺激的脂肪分解在3T3-L1 adipocytes中
机译:甘氨酸抑制脂肪发生,在3T3-L1脂肪细胞中的脂解,巨噬细胞刺激肿瘤坏死因子-α分泌,以及3T3-L1脂肪细胞的炎症介质
机译:姜黄素可减轻肿瘤坏死因子-α刺激的内皮细胞中核因子-κB,c-Jun N-末端激酶和p38的表达
机译:肿瘤坏死因子-α(TNF-α)可能在Duroc×Landrace×Large White生长的手推车的餐后代谢调节中起作用
机译:脂肪特异性蛋白27对肿瘤坏死因子α诱导的人脂肪细胞脂解和胰岛素抵抗的保护作用
机译:迷迭香酸抑制3T3-L1脂肪细胞的脂肪生成脂解巨噬细胞中脂多糖刺激的肿瘤坏死因子-α分泌以及3T3-L1脂肪细胞的炎症介质
机译:肿瘤坏死因子 - 在3T3-L1脂肪细胞中刺激脂肪分解是葡萄糖依赖性:对脂解的长期调节的影响