机译:MRI在经典非同源终端连接期间是一种DNA损伤响应适配器
Department of Pathology and Laboratory Medicine Weill Cornell Medical College;
Department of Pathology and Immunology Washington University School of Medicine;
Department of Pathology and Laboratory Medicine Weill Cornell Medical College;
Department of Pathology and Immunology Washington University School of Medicine;
Department of Pathology and Immunology Washington University School of Medicine;
Immunology Program Memorial Sloan Kettering Cancer Center;
The Department of Molecular Biosciences and the Howard Hughes Medical Institute The University of;
Institute for Cancer Genetics Columbia University College of Physicians and Surgeons;
Department of Pathology and Laboratory Medicine Weill Cornell Medical College;
Department of Pathology and Laboratory Medicine Weill Cornell Medical College;
Department of Pathology and Immunology Washington University School of Medicine;
Department of Chemistry Washington University One Brookings Drive;
The Department of Molecular Biosciences and the Howard Hughes Medical Institute The University of;
Department of Pathology and Laboratory Medicine Weill Cornell Medical College;
Immunology Program Memorial Sloan Kettering Cancer Center;
Molecular Biology Program Memorial Sloan Kettering Cancer Center;
Department of Pathology and Immunology Washington University School of Medicine;
Department of Pathology and Immunology Washington University School of Medicine;
Institute for Cancer Genetics Columbia University College of Physicians and Surgeons;
Department of Pathology and Laboratory Medicine Weill Cornell Medical College;
Department of Pathology and Laboratory Medicine Weill Cornell Medical College;
non-homologous end joining; V(D)J recombination; DNA double-strand break repair; MRI; adaptor protein; CYREN;
机译:MRI在经典非同源终端连接期间是一种DNA损伤响应适配器
机译:裂殖酵母Rad32,Mre11同源物和其他DNA损伤反应蛋白在非同源末端连接和端粒长度维持中的作用
机译:R-Roscovitine(Seliciclib)防止DNA损伤诱导的细胞周期蛋白A1上调并阻碍非同源末端连接(NHEJ)DNA修复
机译:通过原核非同源终端连接途径修复DNA双链断裂
机译:来自多种途径的DNA损伤反应因子介导了替代末端连接。
机译:MRI是经典非同源末端连接过程中的DNA损伤响应适配器
机译:ATM通过在破碎的复制叉时弯曲DNA损伤响应,以对抗毒性无同源终端连接