机译:在普鲁普列南血症中减少MXRA5,并通过MAPK途径影响滋养细胞侵袭
Cangzhou Cent Hosp Dept Obstet 1 Cangzhou 061001 Peoples R China;
Liaocheng Peoples Hosp Dept Gynecol &
Obstet Liaocheng 252000 Peoples R China;
Hebei Gen Hosp Dept Obstet Shijiazhuang 050051 Hebei Peoples R China;
Binzhou City Ctr Hosp Dept Gynaecol Binzhou 251700 Peoples R China;
Jinan Univ Zhengzhou Hosp 2 Affiliated Zhengzhou Hosp Dept Gynaecol 90 Middle Hanghai Rd;
Matrix-remodeling associated 5; Preeclampsia; Trophoblast; Invasion; MAPK;
机译:在普鲁普列南血症中减少MXRA5,并通过MAPK途径影响滋养细胞侵袭
机译:同质酸盐加剧了人类滋养细胞的侵袭并调节了细胞内信号传导途径,包括PI3K / AKT和MAPK途径
机译:在预口度下降并影响人滋养细胞的迁移,侵袭,增殖和管形成
机译:p,p'-DDE对体外培养的大鼠支持细胞细胞内ERK MAPK信号转导途径的影响
机译:正常妊娠和子痫前期人类滋养细胞的分化和侵袭。
机译:miR-30b通过靶向MXRA5抑制胎盘滋养层细胞的活力侵袭和凋亡而促进子痫前期
机译:DSCS表达的CD82通过InflyInbeta1 / Mapk / MapK3 / 1信号传导途径控制滋养细胞的侵袭性,在人类初期妊娠1中