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首页> 外文期刊>Families, systems & health: the journal of collaborative family healthcare >Linking Family and Intimate Partner Relationships to Chronic Pain: An Application of the Biobehavioral Family Model
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Linking Family and Intimate Partner Relationships to Chronic Pain: An Application of the Biobehavioral Family Model

机译:将家庭与慢性痛苦的亲密合作伙伴关系联系起来:生物侵蚀性家族模型的应用

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摘要

Introduction: Research is needed to determine mechanisms of effect linking family relationships and chronic pain for adults. Guided by the Biobehavioral Family Model (BBFM), the present study examined indirect effects between a negative family emotional climate and chronic pain disease activity, as mediated by biobehavioral reactivity. Method: Data for this study are from the Midlife Development in the United States; specifically, a subsample of participants who reported experiencing chronic pain (n = 1,461, ages 32- 84). Participants self-reported family strain, biobehavioral reactivity (i.e., anxiety, depression), and chronic pain disease activity (i.e., pain interference, global health). A subsample of participants (n = 1,070) completed an intimate partner strain measure, indicating they were married/in a committed relationship. Structural equation models were tested with maximum likelihood estimation and bootstrapping. Results: Family strain was indirectly associated with chronic pain disease activity via biobehavioral reactivity-Model 1; chi(2)(10) = 40.75, p < .000, root mean square error of approximation [RMSEA] = .07, comparative fit index [CFI] = .96, standardized root-mean-square residual [SRMR] = .04; partial mediation occurred for partnered participants. This finding was replicated when modeling family strain simultaneously with intimate partner strain, though intimate partner strain was not associated with chronic pain disease activity-Model 2; chi(2)(5) = 8.29, p = .14, RMSEA = .03, CFI = .99, SRMR = .01. Discussion: These findings add to the growing literature that emphasizes the role of family relationships in chronic pain. Future research is needed to replicate our use of the BBFM to specify pathways of effect, incorporating relational and observational data, with diverse samples.
机译:简介:需要研究,以确定将家庭关系和成年人的慢性疼痛联系起来的效果机制。由生物狂欢家庭模型(BBFM)为指导,本研究检测了负面家族情绪气候和慢性疼痛疾病活动之间的间接影响,如生物健康反应性的介导。方法:本研究数据来自美国中期发展;具体而言,报告经历慢性疼痛的参与者的子样本(n = 1,461岁,32-84岁)。与会者自我报告的家庭菌株,生物抗兽反应性(即焦虑,抑郁)和慢性疼痛疾病活动(即疼痛干扰,全球健康)。参与者(n = 1,070)的子样本完成了一个亲密的合作伙伴应变措施,表明他们已婚/以承诺的关系结婚。测试结构方程模型具有最大似然估计和自动启动。结果:系列菌株与慢性疼痛疾病活性通过双向反应性型号1间接相关; Chi(2)(10)= 40.75,p <.000,近似的根均方误差[RMSEA] = .07,比较拟合指数[CFI] = .96,标准化的根均方残留[SRMR] =。 04;合作参与者发生部分调解。尽管亲密的合作伙伴菌株与慢性疼痛疾病活动模型2无关,但在与亲密的合作伙伴菌株同时进行育种菌株时,将复制该发现。 Chi(2)(5)= 8.29,p = .14,RMSEA = .03,CFI = .99,SRMR = .01。讨论:这些调查结果增加了不断增长的文学,强调家庭关系在慢性疼痛中的作用。需要将来的研究复制我们对BBFM的使用,以指定效果的途径,包含多样化的样品。

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