机译:尽管致癌信号堵塞,但代谢重编程确保癌细胞存活
Oregon Hlth &
Sci Univ Knight Comprehens Canc Inst Portland OR 97239 USA;
Oregon Hlth &
Sci Univ Knight Comprehens Canc Inst Portland OR 97239 USA;
Oregon Hlth &
Sci Univ Knight Cardiovasc Inst Portland OR 97239 USA;
Rutgers State Univ Rutgers Canc Inst New Jersey New Brunswick NJ 08903 USA;
Oregon Hlth &
Sci Univ Knight Comprehens Canc Inst Portland OR 97239 USA;
Oregon Hlth &
Sci Univ Knight Comprehens Canc Inst Portland OR 97239 USA;
Oregon Hlth &
Sci Univ Knight Comprehens Canc Inst Portland OR 97239 USA;
Oregon Hlth &
Sci Univ Knight Comprehens Canc Inst Portland OR 97239 USA;
Oregon Hlth &
Sci Univ Knight Comprehens Canc Inst Portland OR 97239 USA;
Oregon Hlth &
Sci Univ Knight Cardiovasc Inst Portland OR 97239 USA;
Oregon Hlth &
Sci Univ Dept Urol Portland OR 97239 USA;
Oregon Hlth &
Sci Univ Dept Urol Portland OR 97239 USA;
Oregon Hlth &
Sci Univ Dept Urol Portland OR 97239 USA;
Univ Calif San Francisco Sch Med Dept Radiol San Francisco CA 94107 USA;
Univ Calif Berkeley Berkeley CA 94720 USA;
Univ Calif Berkeley Berkeley CA 94720 USA;
Univ Calif San Francisco Sch Med Dept Radiol San Francisco CA 94107 USA;
Cardiff Univ Syst Immun Res Inst Cardiff CF14 4XN S Glam Wales;
Univ Calif Berkeley Berkeley CA 94720 USA;
Rutgers State Univ Rutgers Canc Inst New Jersey New Brunswick NJ 08903 USA;
Reed Coll Dept Biol Portland OR 97202 USA;
Oregon Hlth &
Sci Univ Knight Comprehens Canc Inst Portland OR 97239 USA;
autophagy; cancer; metabolism; phospholipid; resistance; signaling;
机译:尽管致癌信号堵塞,但代谢重编程确保癌细胞存活
机译:用穿透细胞的pepducins阻断PAR1信号传导可抑制乳腺癌细胞中的Akt生存途径,并抑制肿瘤的生存和转移。
机译:靶向致癌MUC1-C蛋白可抑制EGFR介导的突变信号和非小细胞肺癌细胞的存活
机译:13C代谢通量癌细胞中AKT信号传导诱导代谢重编程的代谢通量分析
机译:Biochanin A和锰可调节HER-2阳性乳腺癌细胞的致癌信号和抗氧化功能。
机译:代谢重编程可确保癌细胞存活尽管有致癌信号传导阻滞
机译:代谢重编程可确保癌细胞存活,尽管发生了致癌信号传导阻滞