机译:整个转录组测序揭示了婴儿急性髓性白血病的KMT2A-USP2 KMT2A-USP2融合
Department of PediatricsYokohama City University Graduate School of MedicineYokohama Japan;
Department of PediatricsYokohama City University Graduate School of MedicineYokohama Japan;
Department of PediatricsYokohama City University Graduate School of MedicineYokohama Japan;
Department of Pediatric Hematology and Oncology ResearchNational Research Institute for Child;
Department of Maternal‐Fetal BiologyNational Research Institute for Child Health and;
Department of Maternal‐Fetal BiologyNational Research Institute for Child Health and;
Department of Systems BioMedicineNational Research Institute for Child Health and DevelopmentTokyo;
Department of PediatricsYokohama City University Graduate School of MedicineYokohama Japan;
Department of Pediatric Hematology and Oncology ResearchNational Research Institute for Child;
Children's Cancer CenterNational Center for Child Health and DevelopmentTokyo Japan;
Department of Maternal‐Fetal BiologyNational Research Institute for Child Health and;
Department of Pediatric Hematology and Oncology ResearchNational Research Institute for Child;
Department of PediatricsYokohama City University Graduate School of MedicineYokohama Japan;
Department of Pediatric Hematology and Oncology ResearchNational Research Institute for Child;
infant AML; KMT2A; RNA sequencing; USP2;
机译:整个转录组测序揭示了婴儿急性髓性白血病的KMT2A-USP2 KMT2A-USP2融合
机译:隐秘复发acin1 acin1 - Numm1 Nutm1 Numm1融合在非-mt2a kmt2a -rearranged婴儿急性淋巴细胞白血病
机译:一种新型KMT2A-ACTN2 KMT2A-ACTN2婴儿B细胞急性淋巴细胞白血病融合
机译:急性髓性白血病的突畴样素揭示了对白血病的新洞察力
机译:Egr1的丢失在小鼠白血病的发生中起作用,并且可能在急性髓细胞性白血病和与治疗有关的急性髓细胞性白血病的发展中起作用。
机译:11Q23中的治疗诱导缺失导致KMT2a与Arhgef12融合并在治疗的儿童中发育B族急性淋巴相血肿由T(9; 11)(p21; Q23)/ kmt2a-mllt3引起的急性髓性白血病进行治疗
机译:转录组测序显示为急性髓细胞白血病与t(5; 21)(q21; q22)的新型融合伴侣