...
机译:热休克蛋白90 / akt途径参与外源硫化氢对高葡萄糖诱导的H9C2细胞损伤的心脏保护作用(Vol 39,PG 1001,2017)
Shenzhen Sun Yat Sen Cardiovasc Hosp Dept Cardiol Shenzhen Peoples R China;
Third Peoples Hosp Dongguan City Dept Cardiovasc Med Dongguan Peoples R China;
Sun Yat Sen Univ Affiliated Hosp 1 Minist Hlth Dept Cardiol 58 Zhongshan 2rd Rd Guangzhou;
Sun Yat Sen Univ Affiliated Hosp 1 Dept Cardiovasol Guangzhou Guangdong Peoples R China;
Sun Yat Sen Univ Affiliated Hosp 1 Dept Cardiovasol Guangzhou Guangdong Peoples R China;
Sun Yat Sen Univ Affiliated Hosp 1 Minist Hlth Dept Cardiol 58 Zhongshan 2rd Rd Guangzhou;
Sun Yat Sen Univ Affiliated Hosp1 Huangpu Div Dept Anesthesiol Guangzhou Guangdong Peoples R;
Sun Yat Sen Univ Zhongshan Sch Med Dept Physiol Guangzhou Guangdong Peoples R China;
Sun Yat Sen Univ Affiliated Hosp1 Huangpu Div Dept Anesthesiol Guangzhou Guangdong Peoples R;
Sun Yat Sen Univ Affiliated Hosp 1 Minist Hlth Dept Cardiol 58 Zhongshan 2rd Rd Guangzhou;
Sun Yat Sen Univ Sun Yat Sen Mem Hosp Dept Cardiol Guangzhou Guangdong Peoples R China;
机译:热休克蛋白90 / akt途径参与外源硫化氢对高葡萄糖诱导的H9C2细胞损伤的心脏保护作用(Vol 39,PG 1001,2017)
机译:[更正]热休克蛋白90 / Akt通路参与外源性硫化氢对高糖诱导的H9c2?细胞损伤的心脏保护作用
机译:硫化氢通过刺激ATP敏感钾通道/蛋白激酶C /细胞外信号调节激酶/热休克蛋白90途径来保护神经元免受缺氧损伤。
机译:在暴露于植物凝集素后,肠道上皮细胞中的热休克蛋白70和90水平降低
机译:热激蛋白70,热激蛋白90和热激因子1在体外温度和体外及体内PGF2α刺激后在牛黄体中体中的表达。
机译:热休克蛋白90 / Akt途径参与外源性硫化氢对高糖诱导的H9c2细胞损伤的心脏保护作用
机译:热休克蛋白90 / akt途径参与外源硫化氢对高葡萄糖诱导的损伤H9C2细胞的心脏保护作用